PUBLICATION

Colonization of larval zebrafish (Danio rerio) with adherent-invasive Escherichia coli prevents recovery of the intestinal mucosa from drug-induced enterocolitis

Authors
Flores, E., Dutta, S., Bosserman, R., van Hoof, A., Krachler, A.-.M.
ID
ZDB-PUB-231118-2
Date
2023
Source
mSphere   8(6): e0051223 (Journal)
Registered Authors
Keywords
AIEC, colitis, intestinal colonization, zebrafish
MeSH Terms
  • Zebrafish
  • Animals
  • Escherichia coli/genetics
  • Inflammatory Bowel Diseases*
  • Crohn Disease*/complications
  • Humans
  • Enterocolitis*/complications
  • Colitis*/chemically induced
  • Escherichia coli Infections*
  • Intestinal Mucosa
PubMed
37971273 Full text @ mSphere
Abstract
Although inflammatory bowel diseases are on the rise, what factors influence IBD risk and severity, and the underlying mechanisms remain to be fully understood. Although host genetics, microbiome, and environmental factors have all been shown to correlate with the development of IBD, cause and effect are difficult to disentangle in this context. For example, AIEC is a known pathobiont found in IBD patients, but it remains unclear if gut inflammation during IBD facilitates colonization with AIEC, or if AIEC colonization makes the host more susceptible to pro-inflammatory stimuli. It is critical to understand the mechanisms that contribute to AIEC infections in a susceptible host in order to develop successful therapeutics. Here, we show that the larval zebrafish model recapitulates key features of AIEC infections in other animal models and can be utilized to address these gaps in knowledge.
Genes / Markers
Figures
Show all Figures
Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping