PUBLICATION

Paternal hypoxia exposure primes offspring for increased hypoxia resistance

Authors
Ragsdale, A., Ortega-Recalde, O., Dutoit, L., Besson, A.A., Chia, J.H.Z., King, T., Nakagawa, S., Hickey, A., Gemmell, N.J., Hore, T., Johnson, S.L.
ID
ZDB-PUB-220831-15
Date
2022
Source
BMC Biology   20: 185 (Journal)
Registered Authors
Keywords
Cross-generational, DNA methylation, Danio rerio, Epigenetic, Intergenerational, Transcriptomics, Transgenerational acclimation, Transgenerational plasticity, Zebrafish
Datasets
GEO:GSE160662
MeSH Terms
  • Acclimatization
  • Animals
  • Epigenesis, Genetic
  • Humans
  • Hypoxia/genetics
  • Male
  • Paternal Exposure*
  • Zebrafish*/genetics
PubMed
36038899 Full text @ BMC Biol.
Abstract
In a time of rapid environmental change, understanding how the challenges experienced by one generation can influence the fitness of future generations is critically needed. Using tolerance assays and transcriptomic and methylome approaches, we use zebrafish as a model to investigate cross-generational acclimation to hypoxia.
We show that short-term paternal exposure to hypoxia endows offspring with greater tolerance to acute hypoxia. We detected two hemoglobin genes that are significantly upregulated by more than 6-fold in the offspring of hypoxia exposed males. Moreover, the offspring which maintained equilibrium the longest showed greatest upregulation in hemoglobin expression. We did not detect differential methylation at any of the differentially expressed genes, suggesting that other epigenetic mechanisms are responsible for alterations in gene expression.
Overall, our findings suggest that an epigenetic memory of past hypoxia exposure is maintained and that this environmentally induced information is transferred to subsequent generations, pre-acclimating progeny to cope with hypoxic conditions.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping