Morpholino

MO1-rbpms2b

ID
ZDB-MRPHLNO-130820-7
Name
MO1-rbpms2b
Previous Names
None
Target
Sequence
5' - CTTGACACTCATCTTGTGCGTAAAC - 3'
Disclaimer
Although ZFIN verifies reagent sequence data, we recommend that you conduct independent sequence analysis before ordering any reagent.
Note
Translation-blocking MO.
Genome Resources
None
Target Location
Genomic Features
No data available
Expression
Gene expression in Wild Types + MO1-rbpms2b
No data available
Phenotype
Phenotype resulting from MO1-rbpms2b
No data available
Phenotype of all Fish created by or utilizing MO1-rbpms2b
Phenotype Fish Conditions Figures
amacrine cell dendrite decreased branchiness, abnormal WT + MO1-rbpms2a + MO1-rbpms2b standard conditions Fig. 5Fig. 6 from Hörnberg et al., 2013
dorsomedial optic tract axon mislocalised, abnormal WT + MO1-rbpms2a + MO1-rbpms2b control Fig. 1 from Hörnberg et al., 2016
amacrine cell dendritic tree decreased area, abnormal WT + MO1-rbpms2a + MO1-rbpms2b standard conditions Fig. 5Fig. 6 from Hörnberg et al., 2013
dorsomedial optic tract morphology, abnormal WT + MO1-rbpms2a + MO1-rbpms2b control Fig. 1 from Hörnberg et al., 2016
amacrine cell dendrite has fewer parts of type amacrine cell dendritic branch, abnormal WT + MO1-rbpms2a + MO1-rbpms2b standard conditions Fig. 5Fig. 6 from Hörnberg et al., 2013
optomotor response increased process quality, abnormal WT + MO1-rbpms2a + MO1-rbpms2b standard conditions Fig. 8 from Hörnberg et al., 2013
amacrine cell dendrite morphogenesis process quality, abnormal WT + MO1-rbpms2a + MO1-rbpms2b standard conditions Fig. 6 from Hörnberg et al., 2013
amacrine cell dendritic tree decreased length, abnormal WT + MO1-rbpms2a + MO1-rbpms2b standard conditions Fig. 5Fig. 6Fig. 7 from Hörnberg et al., 2013
amacrine cell synapse condensed, abnormal WT + MO1-rbpms2a + MO1-rbpms2b standard conditions Fig. 7 from Hörnberg et al., 2013
amacrine cell dendrite extension process quality, abnormal cu2Tg + MO1-rbpms2a + MO1-rbpms2b standard conditions Fig. 3 from Hörnberg et al., 2013
dorsomedial optic tract morphology, abnormal cu2Tg + MO1-rbpms2a + MO1-rbpms2b control Fig. 4 from Hörnberg et al., 2016
dorsomedial optic tract axon mislocalised, abnormal cu2Tg + MO1-rbpms2a + MO1-rbpms2b control Fig. 4 from Hörnberg et al., 2016
dorsomedial optic tract axon mislocalised, ameliorated cu2Tg + MO1-rbpms2a + MO1-rbpms2b + MO2-nrp1a control Fig. 4 from Hörnberg et al., 2016
dorsomedial optic tract morphology, ameliorated cu2Tg + MO1-rbpms2a + MO1-rbpms2b + MO2-nrp1a control Fig. 4 from Hörnberg et al., 2016
eye ribosome l1camb expression increased amount, abnormal cu8Tg + MO1-rbpms2a + MO1-rbpms2b control Fig. 2 from Hörnberg et al., 2016
eye ribosome alcamb expression increased amount, abnormal cu8Tg + MO1-rbpms2a + MO1-rbpms2b control Fig. 2 from Hörnberg et al., 2016
eye ribosome nrp1a expression increased amount, abnormal cu8Tg + MO1-rbpms2a + MO1-rbpms2b control Fig. 2 from Hörnberg et al., 2016
eye ribosome l1cama expression increased amount, abnormal cu8Tg + MO1-rbpms2a + MO1-rbpms2b control Fig. 2 from Hörnberg et al., 2016
eye ribosome alcama expression increased amount, abnormal cu8Tg + MO1-rbpms2a + MO1-rbpms2b control Fig. 2 from Hörnberg et al., 2016
eye ribosome nrp1b expression increased amount, abnormal cu8Tg + MO1-rbpms2a + MO1-rbpms2b control Fig. 2 from Hörnberg et al., 2016
Citations