Fig. 3
LLC deficit is independent of otolith morphology in chd7 chromodomain mutants. (A) Acoustic startle responses, average short‐latency c‐bend (SLC) frequency comparing chd7 ncu101/+ and chd7 ncu101/ncu101 with or without otolith defects (chd7 +/+ n = 11, chd7 ncu101/+ w/o otolith defect n = 22, chd7 ncu101/+ with otolith defect n = 17, chd7 ncu101/ncu101 w/o otolith defect n = 8, chd7 ncu101/ncu101 with otolith defect n = 11) (mean ± SEM). (B) Short‐latency c‐bend sensitivity index, calculated by the area under the SLC frequency curves for individual larvae (mean ± SD, Ordinary one‐way ANOVA with Tukey's multiple comparison). (C) Long‐latency c‐bend (LLC) frequency as acoustic stimulus intensity increases (mean ± SEM). (D) Long‐latency c‐bend sensitivity index calculated by the area under the LLC frequency curves for individual larvae (mean ± SD, Ordinary one‐way ANOVA with Tukey's multiple comparison, **p < 0.01, ****p < 0.0001).