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Fig. 3
ahr2hu3335 embryos are resistant to TCDD-induced developmental abnormalities.
A) Percent of embryos with axis malformations and B) percent incidence pericardial edema at 120 hpf in embryos treated with 0, 0.1, 1 or 10 nM TCDD from 6–24 hpf. Vehicle control groups (c, 0.1% DMSO) are displayed at 10-4 for graphing purposes. Data represent three independent experiments with 20 embryos per treatment group. C) Representative image of 120 hpf ahr2+ and (D) ahr2hu3335 embryos developmentally exposed to 10 nM TCDD.
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