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Fig. 3 Expression of calsenilin in mutants with affected retinoic acid synthesizing aldehyde dehydrogenase (RALDH; nls) and Notch (mib) signaling. Whole-mount in situ hybridization with digoxigenin-labeled antisense insulin and calsenilin probes on wild-type (WT), nls mutant defective in retinoic acid (RA) synthesis and mib mutants defective in the Delta-Notch pathway. Black and white arrows indicate insulin and calsenilin expression in the pancreas, respectively.
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