FIGURE
            Fig. 2
- ID
- ZDB-FIG-120601-103
- Publication
- He et al., 2012 - The Viral TRAF Protein (ORF111L) from Infectious Spleen and Kidney Necrosis Virus Interacts with TRADD and Induces Caspase 8-mediated Apoptosis
- Other Figures
- All Figure Page
- Back to All Figure Page
                
                    
                        Fig. 2
                    
                    
                
                
            
        
        
    
        
            
            
| ISKNV ORF111L overexpression resulted in abnormal phenotype in zebrafish embryo. (A–B) The GFP and 111L-GFP expression in pEGFP-N3- and p111L-GFP-injected embryos were shown. (C–D) Empty vector pEGFP-N3-injected embryo did not show any phenotypic abnormality, while the yolk sac was not completely developed in 111L-GFP overexpressing embryo. (E–F) Hematoxylin-eosin staining assay was performed in GFP- and 111L-GFP-overexpressing embryo. | 
                
                    
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                        Expression Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Antibody Labeling
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Phenotype Data
                    
                    
                
                
            
        
        
    
        
            
            
            
            
    
    
                
                    
                        Phenotype Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Acknowledgments
                    
                    
                
                
            
        
        
    
        
            
            
                
                    
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      Full text @ PLoS One
                
                
            
        
        
    
    
     
        