CRISPR

CRISPR1-wnt16

ID

ZDB-CRISPR-171004-1

Name

CRISPR1-wnt16

Previous Names

None

Target

wnt16 (1)

Sequence

5' - GTAGGAGTGCCCGAGAAGTT - 3'

Disclaimer

Although ZFIN verifies reagent sequence data, we recommend that you conduct independent sequence analysis before ordering any reagent.

Note

None

Genome Resources

None

Target Location

Constructs

No data available

Genomic Features

Genomic Feature	Affected Genomic Regions
bsl451	wnt16
bsl667	wnt16

Expression

Gene expression in Wild Types + CRISPR1-wnt16

No data available

Phenotype

Phenotype resulting from CRISPR1-wnt16

No data available

Phenotype of all Fish created by or utilizing CRISPR1-wnt16

Phenotype	Fish	Conditions	Figures
caudal fin principal ray bone callus increased amount, abnormal	wnt16^{bsl451/bsl451}	standard conditions	Fig 1 from McGowan et al., 2021
caudal fin principal ray mineral decreased amount, abnormal	wnt16^{bsl451/bsl451}	standard conditions	Fig 1 from McGowan et al., 2021
caudal fin principal ray bone regeneration decreased process quality, abnormal	wnt16^{bsl451/bsl451}	compression injury: caudal fin	Fig 2 from McGowan et al., 2021
caudal fin principal ray fractured, abnormal	wnt16^{bsl451/bsl451}	standard conditions	Fig 1 from McGowan et al., 2021
caudal fin principal ray bone callus increased amount, abnormal	wnt16^{bsl667/bsl667}	standard conditions	Fig 1 from McGowan et al., 2021
caudal fin principal ray mineral decreased amount, abnormal	wnt16^{bsl667/bsl667}	standard conditions	Fig 1 from McGowan et al., 2021
caudal fin principal ray bone regeneration decreased process quality, abnormal	wnt16^{bsl667/bsl667}	compression injury: caudal fin	Fig 2 from McGowan et al., 2021
caudal fin principal ray fractured, abnormal	wnt16^{bsl667/bsl667}	standard conditions	Fig 1 from McGowan et al., 2021
Meckel's cartilage joint morphology, abnormal	hu5910Tg + CRISPR1-wnt16 + CRISPR2-wnt16	standard conditions	Fig. 3 from Brunt et al., 2017
palatoquadrate cartilage joint morphology, abnormal	hu5910Tg + CRISPR1-wnt16 + CRISPR2-wnt16	standard conditions	Fig. 3 from Brunt et al., 2017
caudal fin principal ray bone callus EGFP expression increased amount, abnormal	wnt16^{bsl451/bsl451}; b1212Tg	compression injury: caudal fin	Fig 2 from McGowan et al., 2021
osteoclast acid phosphatase activity increased process quality, abnormal	wnt16^{bsl451/bsl451}; gl23Tg	compression injury: caudal fin	Fig 4 from McGowan et al., 2021
caudal fin principal ray osteoclast increased amount, abnormal	wnt16^{bsl451/bsl451}; gl23Tg	compression injury: caudal fin	Fig 4 from McGowan et al., 2021
lepidotrichium EGFP expression increased amount, abnormal	wnt16^{bsl451/bsl451}; ia4Tg	compression injury: caudal fin	Fig 5 from McGowan et al., 2021
lepidotrichium canonical Wnt signaling pathway increased process quality, abnormal	wnt16^{bsl451/bsl451}; ia4Tg	compression injury: caudal fin	Fig 5 from McGowan et al., 2021
caudal fin principal ray bone callus EGFP expression increased amount, abnormal	wnt16^{bsl667/bsl667}; b1212Tg	compression injury: caudal fin	Fig 2 from McGowan et al., 2021
osteoclast acid phosphatase activity increased process quality, abnormal	wnt16^{bsl667/bsl667}; gl23Tg	compression injury: caudal fin	Fig 4 from McGowan et al., 2021
caudal fin principal ray osteoclast increased amount, abnormal	wnt16^{bsl667/bsl667}; gl23Tg	compression injury: caudal fin	Fig 4 from McGowan et al., 2021
lepidotrichium EGFP expression increased amount, abnormal	wnt16^{bsl667/bsl667}; ia4Tg	compression injury: caudal fin	Fig 5 from McGowan et al., 2021
lepidotrichium canonical Wnt signaling pathway increased process quality, abnormal	wnt16^{bsl667/bsl667}; ia4Tg	compression injury: caudal fin	Fig 5 from McGowan et al., 2021

Citations