CRISPR

CRISPR1-wnt16

ID
ZDB-CRISPR-171004-1
Name
CRISPR1-wnt16
Previous Names
None
Target
Sequence
5' - GTAGGAGTGCCCGAGAAGTT - 3'
Disclaimer
Although ZFIN verifies reagent sequence data, we recommend that you conduct independent sequence analysis before ordering any reagent.
Note
None
Genome Resources
None
Target Location
Constructs
No data available
Genomic Features
Genomic Feature Affected Genomic Regions
bsl451 wnt16
bsl667 wnt16
Expression
Gene expression in Wild Types + CRISPR1-wnt16
No data available
Phenotype
Phenotype resulting from CRISPR1-wnt16
No data available
Phenotype of all Fish created by or utilizing CRISPR1-wnt16
Phenotype Fish Conditions Figures
caudal fin principal ray bone callus increased amount, abnormal wnt16bsl451/bsl451 standard conditions Fig 1 with image from McGowan et al., 2021
caudal fin principal ray mineral decreased amount, abnormal wnt16bsl451/bsl451 standard conditions Fig 1 with image from McGowan et al., 2021
caudal fin principal ray bone regeneration decreased process quality, abnormal wnt16bsl451/bsl451 compression injury: caudal fin Fig 2 with image from McGowan et al., 2021
caudal fin principal ray fractured, abnormal wnt16bsl451/bsl451 standard conditions Fig 1 with image from McGowan et al., 2021
caudal fin principal ray bone callus increased amount, abnormal wnt16bsl667/bsl667 standard conditions Fig 1 with image from McGowan et al., 2021
caudal fin principal ray mineral decreased amount, abnormal wnt16bsl667/bsl667 standard conditions Fig 1 with image from McGowan et al., 2021
caudal fin principal ray bone regeneration decreased process quality, abnormal wnt16bsl667/bsl667 compression injury: caudal fin Fig 2 with image from McGowan et al., 2021
caudal fin principal ray fractured, abnormal wnt16bsl667/bsl667 standard conditions Fig 1 with image from McGowan et al., 2021
Meckel's cartilage joint morphology, abnormal hu5910Tg + CRISPR1-wnt16 + CRISPR2-wnt16 standard conditions Fig. 3 with image from Brunt et al., 2017
palatoquadrate cartilage joint morphology, abnormal hu5910Tg + CRISPR1-wnt16 + CRISPR2-wnt16 standard conditions Fig. 3 with image from Brunt et al., 2017
caudal fin principal ray bone callus EGFP expression increased amount, abnormal wnt16bsl451/bsl451; b1212Tg compression injury: caudal fin Fig 2 with image from McGowan et al., 2021
osteoclast acid phosphatase activity increased process quality, abnormal wnt16bsl451/bsl451; gl23Tg compression injury: caudal fin Fig 4 with image from McGowan et al., 2021
caudal fin principal ray osteoclast increased amount, abnormal wnt16bsl451/bsl451; gl23Tg compression injury: caudal fin Fig 4 with image from McGowan et al., 2021
lepidotrichium EGFP expression increased amount, abnormal wnt16bsl451/bsl451; ia4Tg compression injury: caudal fin Fig 5 with image from McGowan et al., 2021
lepidotrichium canonical Wnt signaling pathway increased process quality, abnormal wnt16bsl451/bsl451; ia4Tg compression injury: caudal fin Fig 5 with image from McGowan et al., 2021
caudal fin principal ray bone callus EGFP expression increased amount, abnormal wnt16bsl667/bsl667; b1212Tg compression injury: caudal fin Fig 2 with image from McGowan et al., 2021
osteoclast acid phosphatase activity increased process quality, abnormal wnt16bsl667/bsl667; gl23Tg compression injury: caudal fin Fig 4 with image from McGowan et al., 2021
caudal fin principal ray osteoclast increased amount, abnormal wnt16bsl667/bsl667; gl23Tg compression injury: caudal fin Fig 4 with image from McGowan et al., 2021
lepidotrichium EGFP expression increased amount, abnormal wnt16bsl667/bsl667; ia4Tg compression injury: caudal fin Fig 5 with image from McGowan et al., 2021
lepidotrichium canonical Wnt signaling pathway increased process quality, abnormal wnt16bsl667/bsl667; ia4Tg compression injury: caudal fin Fig 5 with image from McGowan et al., 2021
Citations