PUBLICATION

3D spatial organization of heterogeneous nkx2.5+ progenitors in the zebrafish heart field pre-patterns cardiovascular development

Authors
Coskun, H., Wang, Y., Gao, X., Chen, K., Geoffrey Burns, C., Burns, C.E.
ID
ZDB-PUB-251126-11
Date
2025
Source
Nature communications : (Journal)
Registered Authors
Burns (Erter), Caroline
Keywords
none
Datasets
GEO:GSE294469, GEO:GSE294086
MeSH Terms
  • Animals
  • Branchial Region/embryology
  • Cell Movement
  • Gene Expression Regulation, Developmental
  • Heart*/embryology
  • Homeobox Protein Nkx-2.5*/genetics
  • Homeobox Protein Nkx-2.5*/metabolism
  • Myocardium/cytology
  • Myocardium/metabolism
  • Myocytes, Cardiac/cytology
  • Myocytes, Cardiac/metabolism
  • Stem Cells*/cytology
  • Stem Cells*/metabolism
  • Zebrafish*/embryology
  • Zebrafish*/genetics
  • Zebrafish*/metabolism
  • Zebrafish Proteins*/genetics
  • Zebrafish Proteins*/metabolism
PubMed
41290702 Full text @ Nat. Commun.
Abstract
Nkx2.5+ progenitor cells are specified in the bilateral zebrafish heart field, which contributes to multiple cardiovascular derivatives. After specification, the field physically separates into a cardiac fraction that migrates to the midline to form ventricular and atrial myocardium, a pharyngeal fraction that is sequestered in the pharyngeal arches before generating head muscles, the outflow tract, and pharyngeal arch arteries, and a lateral fraction that becomes inflow atrial and pacemaker cardiomyocytes as well as pericardium. Whether these progenitors are transcriptionally distinct and regionally organized has remained unclear. Here, we demonstrate that the heart field is composed of heterogeneous nkx2.5+ progenitor populations that are arranged according to their migratory paths and end fates. Through prospective fate mapping and mutant analyses, we define the derivatives of each subgroup. Our data provide a 3D blueprint of heart field organization, offering insights into cardiovascular development and identifying vulnerabilities contributing to congenital heart disease.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping