PUBLICATION

MEF2C controls segment-specific gene regulatory networks that direct heart tube morphogenesis

Authors
Muncie-Vasic, J.M., Sinha, T., Clark, A.P., Brower, E.F., Saucerman, J.J., Black, B.L., Bruneau, B.G.
ID
ZDB-PUB-250830-10
Date
2025
Source
Genes & Development : (Journal)
Registered Authors
Black, Brian
Keywords
cardiogenesis, developmental biology, embryogenesis, gene regulation, gene regulatory networks, heart development
MeSH Terms
  • Animals
  • Enhancer Elements, Genetic
  • Gene Expression Regulation, Developmental*
  • Gene Regulatory Networks*/genetics
  • Heart*/embryology
  • MEF2 Transcription Factors*/genetics
  • MEF2 Transcription Factors*/metabolism
  • Morphogenesis*/genetics
  • Zebrafish*/embryology
  • Zebrafish*/genetics
  • Zebrafish Proteins*/genetics
  • Zebrafish Proteins*/metabolism
PubMed
40883017 Full text @ Genes & Dev.
Abstract
The gene regulatory networks (GRNs) that control early heart formation are beginning to be understood, but lineage-specific GRNs remain largely undefined. We investigated networks controlled by the vital transcription factor MEF2C using a time course of single-nucleus RNA sequencing and ATAC sequencing in wild-type and Mef2c-null embryos. We identified a "posteriorized" cardiac gene signature and chromatin landscape in the absence of MEF2C. Integrating our multiomics data in a deep learning-based model, we constructed developmental trajectories for each of the outflow tract, ventricular, and inflow tract segments and alterations of these in Mef2c-null embryos. We computationally identified segment-specific MEF2C-dependent enhancers with activity in the developing zebrafish heart. Finally, using inferred GRNs, we discovered that the Mef2c-null heart malformations are partly driven by increased activity of the nuclear hormone receptor NR2F2. Our results delineate lineage-specific GRNs in the early heart tube and provide a generalizable framework for dissecting transcriptional networks governing developmental processes.
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