PUBLICATION

Evaluation of Phenazine Derivatives from the Lichen-Associated Streptomyces flavidovirens as Potent Antineuroinflammatory Agents In Vitro and In Vivo

Authors
Gao, L.L., Gao, Y.Q., Liu, W.Y., Stadler, M., Zhu, Y.T., Qi, J.Z., Han, W.B., Gao, J.M.
ID
ZDB-PUB-240815-1
Date
2024
Source
Journal of natural products   87(8): 1930-1940 (Journal)
Registered Authors
Keywords
none
MeSH Terms
  • Animals
  • Lipopolysaccharides/pharmacology
  • Mice
  • Zebrafish*
  • Microglia/drug effects
  • Signal Transduction/drug effects
  • Molecular Structure
  • Tumor Necrosis Factor-alpha/metabolism
  • Lichens*/chemistry
  • Anti-Inflammatory Agents*/chemistry
  • Anti-Inflammatory Agents*/pharmacology
  • NF-E2-Related Factor 2/metabolism
  • NF-kappa B*/metabolism
  • Phenazines*/chemistry
  • Phenazines*/pharmacology
  • Oxidative Stress/drug effects
  • Streptomyces*/chemistry
PubMed
39140432 Full text @ J. Nat. Prod.
Abstract
Eighteen nitrogen-containing compounds (1-18) were isolated from cultures of the lichen-associated Streptomyces flavidovirens collected from the Qinghai-Tibet Plateau, including seven phenazine derivatives with three new ones, named subphenazines A-C (2-4), two new furan pyrrolidones (8-9), and nine known alkaloids. The structures were elucidated by spectroscopic data analysis, and absolute configurations were determined by single-crystal X-ray diffraction and ECD calculations. The phenazine-type derivatives, in particular compound 3, exhibited significantly better antineuroinflammatory activity than other isolated compounds (8-18). Compound 3 inhibited the release of proinflammatory cytokines including IL-6, TNF-α, and PGE2, and the nuclear translocation of NF-κB; it also reduced the oxidative stress and activated the Nrf2 signaling pathway in LPS-induced BV2 microglia cells. In vivo anti-inflammatory activity in zebrafish indicated that 3 inhibited LPS-stimulated ROS generation. These findings suggested that compound 3 might be a potent antineuroinflammatory agent through the regulation of the NF-κB/Nrf2 signaling pathways.
Genes / Markers
Figures
Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping