PUBLICATION

Development of a hepatic cryoinjury model to study liver regeneration

Authors
Sande-Melon, M., Bergemann, D., Fernández-Lajarín, M., González-Rosa, J.M., Cox, A.G.
ID
ZDB-PUB-240801-1
Date
2024
Source
Development (Cambridge, England)   151(15): (Journal)
Registered Authors
Cox, Andrew
Keywords
Apoptosis, Cryoinjury, Fibrosis, Inflammation, Liver regeneration, Necrosis, Proliferation, Zebrafish
Datasets
GEO:GSE245878
MeSH Terms
  • Animals
  • Apoptosis
  • Disease Models, Animal
  • Liver*/metabolism
  • Liver*/pathology
  • Liver Cirrhosis/pathology
  • Liver Regeneration*/physiology
  • Male
  • Mice
  • Mice, Inbred C57BL
PubMed
38975841 Full text @ Development
Abstract
The liver is a remarkable organ that can regenerate in response to injury. Depending on the extent of injury, the liver can undergo compensatory hyperplasia or fibrosis. Despite decades of research, the molecular mechanisms underlying these processes are poorly understood. Here, we developed a new model to study liver regeneration based on cryoinjury. To visualise liver regeneration at cellular resolution, we adapted the CUBIC tissue-clearing approach. Hepatic cryoinjury induced a localised necrotic and apoptotic lesion characterised by inflammation and infiltration of innate immune cells. After this initial phase, we observed fibrosis, which resolved as regeneration re-established homeostasis in 30 days. Importantly, this approach enables the comparison of healthy and injured parenchyma within an individual animal, providing unique advantages to previous models. In summary, the hepatic cryoinjury model provides a fast and reproducible method for studying the cellular and molecular pathways underpinning fibrosis and liver regeneration.
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