PUBLICATION

Aerobic glycolysis is important for zebrafish larval wound closure and tail regeneration

Authors
Scott, C.A., Carney, T.J., Amaya, E.
ID
ZDB-PUB-220924-33
Date
2022
Source
Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society   30(6): 665-680 (Journal)
Registered Authors
Amaya, Enrique, Carney, Tom
Keywords
Laconic, Warburg Effect, appendage regeneration, lactate, metabolism, wound healing
MeSH Terms
  • Animals
  • Glycolysis
  • Humans
  • Lactates/pharmacology
  • Larva
  • Regeneration/physiology
  • Wound Healing*/physiology
  • Zebrafish*/physiology
PubMed
36148505 Full text @ Wound Repair Regen.
Abstract
The underlying mechanisms of appendage regeneration remain largely unknown and uncovering these mechanisms in capable organisms has far-reaching implications for potential treatments in humans. Recent studies implicate a requirement for metabolic reprogramming reminiscent of the Warburg effect during successful appendage and organ regeneration. As changes are thus predicted to be highly dynamic, methods permitting direct, real-time visualization of metabolites at the tissue and organismal level, would offer a significant advance in defining the influence of metabolism on regeneration and healing. We sought to examine whether glycolytic activity was altered during larval fin regeneration, utilising the genetically encoded biosensor, Laconic, enabling the spatiotemporal assessment of lactate levels in living zebrafish. We present evidence for a rapid increase in lactate levels within minutes following injury, with a role of aerobic glycolysis in actomyosin contraction and wound closure. We also find a second wave of lactate production, associated with overall larval tail regeneration. Chemical inhibition of glycolysis attenuates both contraction of the wound and regrowth of tissue following tail amputation, suggesting aerobic glycolysis is necessary at two distinct stages of regeneration.
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