PUBLICATION

Generation and Characterization of a Zebrafish IL-2Rγc SCID Model

Authors
Sertori, R., Jones, R., Basheer, F., Rivera, L., Dawson, S., Loke, S., Heidary, S., Dhillon, A., Liongue, C., Ward, A.C.
ID
ZDB-PUB-220227-11
Date
2022
Source
International Journal of Molecular Sciences   23(4): (Journal)
Registered Authors
Liongue, Clifford, Ward, Alister C.
Keywords
IL-2Rγc, immunodeficiency, microbiota, xenotransplantation, zebrafish
MeSH Terms
  • Amino Acid Sequence
  • Animals
  • Base Sequence
  • Gastrointestinal Microbiome/physiology
  • Interleukin Receptor Common gamma Subunit
  • Killer Cells, Natural/metabolism
  • Lymphopoiesis/physiology
  • Models, Animal
  • Phenotype
  • Severe Combined Immunodeficiency/metabolism*
  • T-Lymphocytes/metabolism
  • Zebrafish/metabolism*
PubMed
35216498 Full text @ Int. J. Mol. Sci.
Abstract
The IL-2 family of cytokines act via receptor complexes that share the interleukin-2 receptor gamma common (IL-2Rγc) chain to play key roles in lymphopoiesis. Inactivating IL-2Rγc mutations results in severe combined immunodeficiency (SCID) in humans and other species. This study sought to generate an equivalent zebrafish SCID model. The zebrafish il2rga gene was targeted for genome editing using TALENs and presumed loss-of-function alleles analyzed with respect to immune cell development and impacts on intestinal microbiota and tumor immunity. Knockout of zebrafish Il-2rγc.a resulted in a SCID phenotype, including a significant reduction in T cells, with NK cells also impacted. This resulted in dysregulated intestinal microbiota and defective immunity to tumor xenotransplants. Collectively, this establishes a useful zebrafish SCID model.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping