PUBLICATION

New Pqs Quorum Sensing System Inhibitor as an Antibacterial Synergist against Multidrug-Resistant Pseudomonas aeruginosa

Authors
Liu, J., Hou, J.S., Chang, Y.Q., Peng, L.J., Zhang, X.Y., Miao, Z.Y., Sun, P.H., Lin, J., Chen, W.M.
ID
ZDB-PUB-220208-1
Date
2022
Source
Journal of medicinal chemistry   65: 688-709 (Journal)
Registered Authors
Keywords
none
MeSH Terms
  • Colony Count, Microbial
  • Animals
  • Pseudomonas Infections/microbiology
  • Pseudomonas aeruginosa/drug effects*
  • Cell Line
  • Pyridines/chemical synthesis
  • Pyridines/pharmacology
  • Pyocyanine/antagonists & inhibitors
  • Drug Resistance, Multiple, Bacterial/drug effects*
  • Zebrafish
  • Quinolones/metabolism
  • Biofilms/drug effects
  • Drug Synergism
  • Quorum Sensing/drug effects*
  • Cell Survival/drug effects
  • Anti-Bacterial Agents/pharmacology*
  • Microbial Sensitivity Tests
PubMed
34951310 Full text @ J. Med. Chem.
Abstract
Development of new bacterial biofilm inhibitors as antibacterial synergists is an effective strategy to solve the resistance of Pseudomonas aeruginosa. In this paper, a series of 3-hydroxy-pyridin-4(1H)-ones were synthesized and evaluated, and the hit compound (20p) was identified with the effects of inhibiting the production of pyocyanin (IC50 = 8.6 μM) and biofilm formation (IC50 = 4.5 μM). Mechanistic studies confirmed that 20p inhibits the formation of bacterial biofilm by inhibiting the expression of pqsA, blocking pqs quorum sensing system quinolone biosynthesis. Moreover, we systematically investigated the bactericidal effects of combining currently approved antibiotics for CF including tobramycin, ciprofloxacin, and colistin E with 20p, which showed obvious antibacterial synergy to overcome antibiotics resistance in multidrug-resistant P. aeruginosa biofilms. The result indicates that compound 20p may be used in the future as a potentially novel antibacterial synergist candidate for the treatment of P. aeruginosa infections.
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