PUBLICATION

Precise base editing for the in vivo study of developmental signaling and human pathologies in zebrafish

Authors
Rosello, M., Vougny, J., Czarny, F., Mione, M.C., Concordet, J.P., Albadri, S., Del Bene, F.
ID
ZDB-PUB-210213-18
Date
2021
Source
eLIFE   10: (Journal)
Registered Authors
Del Bene, Filippo, Mione, Marina, Rosello, Marion
Keywords
developmental biology, genetics, genomics, zebrafish
MeSH Terms
  • Animals
  • Disease Models, Animal
  • Gene Editing
  • Humans
  • Mutation
  • Oncogenes*
  • Point Mutation*
  • Signal Transduction*
  • Zebrafish/metabolism
  • Zebrafish Proteins/genetics*
  • Zebrafish Proteins/metabolism
  • beta Catenin/genetics*
  • beta Catenin/metabolism
PubMed
33576334 Full text @ Elife
Abstract
While zebrafish is emerging as a new model system to study human diseases, an efficient methodology to generate precise point mutations at high efficiency is still lacking. Here we show that base editors can generate C-to-T point mutations with high efficiencies without other unwanted on-target mutations. In addition, we established a new editor variant recognizing an NAA PAM, expanding the base editing possibilities in zebrafish. Using these approaches, we first generated a base change in the ctnnb1 gene, mimicking oncogenic mutations of the human gene known to result in constitutive activation of endogenous Wnt signaling. Additionally, we precisely targeted several cancer-associated genes including cbl. With this last target we created a new zebrafish dwarfism model. Together our findings expand the potential of zebrafish as a model system allowing new approaches for the endogenous modulation of cell signaling pathways and the generation of precise models of human genetic disease associated-mutations.
Genes / Markers
Figures
Show all Figures
Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping