PUBLICATION

Zebrafish brain RNA sequencing reveals that cell adhesion molecules are critical in brain aging

Authors
Erbaba, B., Burhan, Ö.P., Şerifoğlu, N., Muratoğlu, B., Kahveci, F., Adams, M.M., Arslan-Ergül, A.
ID
ZDB-PUB-200708-16
Date
2020
Source
Neurobiology of aging   94: 164-175 (Journal)
Registered Authors
Keywords
Brain aging, Cell adhesion molecules, RNA sequencing, Zebrafish
Datasets
GEO:GSE133436
MeSH Terms
  • Activated-Leukocyte Cell Adhesion Molecule/genetics
  • Activated-Leukocyte Cell Adhesion Molecule/metabolism
  • Aging/genetics*
  • Aging/metabolism*
  • Alzheimer Disease/genetics
  • Alzheimer Disease/metabolism
  • Animals
  • Antigens, CD/genetics
  • Antigens, CD/metabolism
  • Brain/metabolism*
  • Cell Adhesion Molecules/genetics*
  • Cell Adhesion Molecules/metabolism*
  • Cell Adhesion Molecules, Neuronal/genetics
  • Cell Adhesion Molecules, Neuronal/metabolism
  • Connexins/genetics
  • Fetal Proteins/genetics
  • Fetal Proteins/metabolism
  • Gene Expression/genetics
  • Gene Expression Regulation, Developmental/genetics
  • Humans
  • RNA/genetics*
  • RNA/metabolism*
  • Sequence Analysis, RNA/methods*
  • Zebrafish
PubMed
32629311 Full text @ Neurobiol. Aging
Abstract
Brain aging is a complex process, which involves multiple pathways including various components from cellular to molecular. This study aimed to investigate the gene expression changes in zebrafish brains through young-adult to adult, and adult to old age. RNA sequencing was performed on isolated neuronal cells from zebrafish brains. The cells were enriched in progenitor cell markers, which are known to diminish throughout the aging process. We found 176 statistically significant, differentially expressed genes among the groups, and identified a group of genes based on gene ontology descriptions, which were classified as cell adhesion molecules. The relevance of these genes was further tested in another set of zebrafish brains, human healthy, and Alzheimer's disease brain samples, as well as in Allen Brain Atlas data. We observed that the expression change of 2 genes, GJC2 and ALCAM, during the aging process was consistent in all experimental sets. Our findings provide a new set of markers for healthy brain aging and suggest new targets for therapeutic approaches to neurodegenerative diseases.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping