PUBLICATION

Glycine Regulates Neural Stem Cell Proliferation During Development via Lnx1-Dependent Notch Signaling

Authors
Bekri, A., Liao, M., Drapeau, P.
ID
ZDB-PUB-190306-5
Date
2019
Source
Frontiers in molecular neuroscience   12: 44 (Journal)
Registered Authors
Drapeau, Pierre
Keywords
LNX1, NSCs, Notch activity, glycine signaling, neurogenesis
MeSH Terms
none
PubMed
30833887 Full text @ Front. Mol. Neurosci.
Abstract
During development of the zebrafish embryo, glycine signaling promotes the differentiation of neural stem cells (NSCs). We found that glycine signaling suppresses the expression of Ligand of Numb X1 (lnx1, Ligand of numb protein-x1), a gene of unknown function during NSC differentiation that is selectively expressed in the embryonic central nervous system (CNS). As a consequence, Numb levels were stabilized and Notch activity (measured as her4.1 expression) was reduced, promoting NSC differentiation. These consequent actions were blocked by knockdown of lnx1. In contrast, lnx1 overexpression increased NSC proliferation and led to defects of neural tube closure at the early stages of development. Thus, our data provide evidence that glycine/lnx1 signaling modulates NSC proliferation by regulation of Notch signaling.
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