PUBLICATION

Lipid droplet biology and evolution illuminated by the characterization of a novel Perilipin in teleost fish

Authors
Granneman, J.G., Kimler, V.A., Zhang, H., Ye, X., Luo, X., Postlethwait, J.H., Thummel, R.
ID
ZDB-PUB-170301-2
Date
2017
Source
eLIFE   6: (Journal)
Registered Authors
Luo, Xixia, Postlethwait, John H., Thummel, Ryan
Keywords
cell biology, evolutionary biology, genomics, zebrafish
MeSH Terms
  • Animals
  • Carotenoids/metabolism
  • Cyclic AMP-Dependent Protein Kinases/metabolism
  • Gene Expression Profiling
  • Gene Knockout Techniques
  • Lipid Droplets/metabolism*
  • Perilipin-1/genetics
  • Perilipin-1/metabolism*
  • Phosphorylation
  • Protein Processing, Post-Translational
  • Zebrafish*
PubMed
28244868 Full text @ Elife
Abstract
Perilipin (PLIN) proteins constitute an ancient family important in lipid droplet (LD) formation and triglyceride metabolism. We identified an additional PLIN clade (plin6) that is unique to teleosts and can be traced to the two whole genome duplications that occurred early in vertebrate evolution. Plin6 is highly expressed in skin xanthophores, which mediate red/yellow pigmentation and trafficking, but not in tissues associated with lipid metabolism. Biochemical and immunochemical analyses demonstrate that zebrafish Plin6 protein targets the surface of pigment-containing carotenoid droplets (CD). Protein kinase A (PKA) activation, which mediates CD dispersion in xanthophores, phosphorylates Plin6 on conserved residues. Knockout of plin6 in zebrafish severely impairs the ability of CD to concentrate carotenoids and prevents tight clustering of CD within carotenoid bodies. Ultrastructural and functional analyses indicate that LD and CD are homologous structures, and that Plin6 was functionalized early in vertebrate evolution for concentrating and trafficking pigment.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping