PUBLICATION

Knockdown of zebrafish Nanog increases proliferation of primordial germ cells during early embryonic development

Authors
Wang, H., Liu, Y., Ye, D., Li, J., Liu, J., Deng, F.
ID
ZDB-PUB-160430-11
Date
2016
Source
Development, growth & differentiation   58(4): 355-66 (Journal)
Registered Authors
Ye, Ding
Keywords
PGCs, embryonic development, proliferation, zNanog, zebrafish
MeSH Terms
  • Animals
  • Embryo, Nonmammalian/cytology
  • Embryo, Nonmammalian/embryology*
  • Embryonic Development/physiology*
  • Gene Knockdown Techniques
  • Germ Cells/metabolism*
  • Nanog Homeobox Protein/genetics
  • Nanog Homeobox Protein/metabolism*
  • Zebrafish/embryology*
  • Zebrafish Proteins/genetics
  • Zebrafish Proteins/metabolism*
PubMed
27125179 Full text @ Dev. Growth Diff.
Abstract
Nanog is a homeodomain transcription factor that plays a prominent role in maintaining the pluripotency and self-renewal capacity of embryonic stem cells (ESCs) in mammals. Medaka Nanog is necessary for S-phase transition and proliferation during embryonic development. However, whether Nanog regulates the proliferation of primordial germ cells (PGCs) during embryonic development has not yet been investigated. In this study, we identified the homologue of the mammalian Nanog gene in zebrafish (zNanog). The expression of both zNanog mRNA and protein was demonstrated in the spermatogonia (male germ stem cells) of the testis and the early oocytes of the ovary. During the embryonic development, zNanog mRNA is expressed in the cytoplasm of PGCs, and its protein is localized to the PGC nuclei. We also found that zNanog depletion using morpholinos resulted in the increases and aberrant localization of PGCs in the zebrafish embryos from the sphere stage to the 50% epiboly stage. These data indicated that zNanog inhibits the PGCs proliferation in early embryonic development of zebrafish.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping