PUBLICATION

Ribonuclease like 5 regulates zebrafish yolk extension by suppressing a p53-dependent DNA damage response pathway

Authors
Zhai, Y., Zhao, X., Sheng, J., Gao, X., Ou, Z., Xu, Z.
ID
ZDB-PUB-150520-15
Date
2015
Source
The international journal of biochemistry & cell biology   65: 12-19 (Journal)
Registered Authors
Keywords
DNA damage, Embryogenesis, P53, Ribonuclease like 5, Yolk extension
MeSH Terms
  • Animals
  • DNA Damage*
  • Embryo, Nonmammalian
  • Embryonic Development/physiology
  • Gene Knockdown Techniques
  • Ribonuclease, Pancreatic/genetics
  • Ribonuclease, Pancreatic/metabolism*
  • Tumor Suppressor Protein p53/genetics*
  • Tumor Suppressor Protein p53/metabolism
  • Yolk Sac/growth & development
  • Yolk Sac/metabolism
  • Yolk Sac/physiology*
  • Zebrafish/physiology*
  • Zebrafish Proteins/genetics*
  • Zebrafish Proteins/metabolism
PubMed
25980932 Full text @ Int. J. Biochem. Cell Biol.
Abstract
Ribonuclease like 5 (Rnasel5) is a novel member of the zebrafish ribonuclease A family and its expression is increased during early embryogenesis. However, the in vivo biological function of Rnasel5 remains to be elucidated. Here, we report that knockdown of Rnasel5 by morhpolinos caused shrunken yolk extension as well as increased DNA damage at yolk syncytial layer and external tissue layers via the activation of p53 pathway. In addition, the morphological defects caused by Rnasel5 knockdown can be partially rescued by mRNA injection. Our findings provide the first functional characterization of Rnasel5 in zebrafish development and reveal its critical role in yolk extension by modulation of the p53 pathway.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping