PUBLICATION
Correction: Structural and functional properties of ryanodine receptor type 3 in zebrafish tail muscle
- Authors
- Perni, S., Marsden, K.C., Escobar, M., Hollingworth, S., Baylor, S.M., Franzini-Armstrong, C.
- ID
- ZDB-PUB-150408-17
- Date
- 2015
- Source
- The Journal of general physiology 145: 253 (Other)
- Registered Authors
- Marsden, Kurt
- Keywords
- none
- MeSH Terms
- none
- PubMed
- 25712018 Full text @ J. Gen. Physiol.
Citation
Perni, S., Marsden, K.C., Escobar, M., Hollingworth, S., Baylor, S.M., Franzini-Armstrong, C. (2015) Correction: Structural and functional properties of ryanodine receptor type 3 in zebrafish tail muscle. The Journal of general physiology. 145:253.
Abstract
In the original online early release version of this article, the text contained the following three errors. In the last sentence of the section Protein expression silencing in Materials and methods, Fig. S2 should have been cited instead of Fig. S1. In the legend to Fig. 4, the second to last sentence should have said “bin widths of 0.1 sarcomere1 s1” instead of “0.05 sarcomere1 s1.” And in the last sentence of that legend, it should have said “0.05 sarcomere1 s1” instead of “0.025 sarcomere1 s1.” The HTML, PDF, and print versions now appear correctly. For reference, the corrected sentences follow:
The second control was designed to block the Tbx5 protein, which is important in vertebrate limb development (Tamura et al., 1999), allowing us to easily test the efficiency and specificity of the injection by verifying the absence of pectoral fins in morphant larvae (see Fig. S2) and the unaffected presence of PJF.
For frequencies above 0, the symbols are plotted at the center frequency of bin widths of 0.1 sarcomere1 s1. In the case of the morphant cells, sparks were sufficiently rare that the mean value of the frequencies in the first bin is 0.0065 ± 0.0028 sarcomere1 s1 (n = 8; ±SEM), considerably less than the position of the central frequency (0.05 sarcomere1 s1).
Errata / Notes
Correction to ZDB-PUB-150211-4.
Genes / Markers
Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Orthology
Engineered Foreign Genes
Mapping