PUBLICATION

Structural Insights into the C1q Domain of Caprin-2 in Canonical Wnt Signaling

Authors
Miao, H., Jia, Y., Xie, S., Wang, X., Zhao, J., Chu, Y., Zhou, Z., Shi, Z., Song, X., Li, L.
ID
ZDB-PUB-141022-6
Date
2014
Source
The Journal of biological chemistry   289(49): 34104-13 (Journal)
Registered Authors
Keywords
C1q, Caprin-2, Wnt pathway, Wnt signaling, calcium-binding protein, crystal structure, mutagenesis
MeSH Terms
  • Animals
  • Calcium/chemistry*
  • Calcium/metabolism
  • Cell Cycle Proteins/chemistry*
  • Cell Cycle Proteins/genetics
  • Cell Cycle Proteins/metabolism
  • Complement C1q/chemistry*
  • Complement C1q/metabolism
  • Crystallography, X-Ray
  • Escherichia coli/genetics
  • Escherichia coli/metabolism
  • Gene Expression Regulation
  • HEK293 Cells
  • Humans
  • Models, Molecular
  • Mutation
  • Protein Multimerization
  • Protein Structure, Secondary
  • Protein Structure, Tertiary
  • Recombinant Proteins/chemistry
  • Recombinant Proteins/genetics
  • Recombinant Proteins/metabolism
  • Wnt Signaling Pathway
  • Zebrafish
PubMed
25331957 Full text @ J. Biol. Chem.
Abstract
Previously, we have identified Caprin-2 as a new regulator in canonical Wnt signaling through a mechanism of facilitating LRP5/6 phosphorylation; moreover, we found that its C-terminal C1q-related domain (Cap2_CRD) is required for this process. Here, we determined the crystal structures of Cap2_CRD from human and zebrafish, which both associate as a homotrimer with calcium located at the symmetric center. Surprisingly, the calcium- binding-deficient mutant exists as a more stable trimer than its wild-type counterpart. Further studies showed that this Caprin-2 mutant disabled in binding calcium maintains the activity of promoting LRP5/6 phosphorylation; while the mutations disrupting Cap2_CRD homotrimer did impair such activity. Together, our findings suggested that the C-terminal CRD domain of Caprin-2 forms a flexible homotrimer mediated by calcium; and that such trimeric assembly is required for Caprin-2 to regulate canonical Wnt signaling.
Genes / Markers
Figures
Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping