PUBLICATION

An Emerging Water Contaminant, Semicarbazide, Exerts an Anti-estrogenic Effect in Zebrafish (Danio rerio)

Authors
Gao, S., Wang, W., Tian, H., Zhang, X., Guo, L., Ru, S.
ID
ZDB-PUB-140616-1
Date
2014
Source
Bulletin of environmental contamination and toxicology   93(3): 280-8 (Journal)
Registered Authors
Keywords
none
MeSH Terms
  • Zebrafish/metabolism
  • Female
  • Receptors, Estrogen/metabolism*
  • Semicarbazides/toxicity*
  • Endocrine Disruptors/metabolism
  • Endocrine Disruptors/toxicity*
  • Animals
  • Estrogens/metabolism
  • Male
  • Liver/metabolism
  • Estradiol/metabolism
  • Vitellogenins/metabolism*
  • Water Pollutants, Chemical/metabolism
  • Water Pollutants, Chemical/toxicity*
PubMed
24929547 Full text @ Bull. Environ. Contam. Toxicol.
Abstract
To determine the endocrine disrupting effect of semicarbazide, an emerging water contaminant, the changes in transcript levels of hepatic estrogen-response genes including vitellogenin-1 (vtg-1), estrogen receptor α (ERα), and estrogen receptor β (ERβ) were measured in male and female zebrafish exposed to semicarbazide with or without exogenous 17β-estradiol (E2). Exposure of male zebrafish to semicarbazide for 96 h or 28 days resulted in no significant induction in hepatic vtg-1, ERα, or ERβ mRNA expression, indicating that semicarbazide has no estrogenic effect. However, a remarkable anti-estrogenic effect of semicarbazide was demonstrated: semicarbazide treatment of female zebrafish for 96 h and 28 days resulted in significant decreases in transcript levels of vtg-1, ERα, and ERβ, as well as decreases in the gonadosomatic index level after 28 days. Moreover, semicarbazide exposure significantly inhibited the induction of vtg-1, ERα and ERβ mRNA by E2 when male zebrafish were co-exposed for 28 days.
Genes / Markers
Figures
Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping