PUBLICATION

Structure of the caspase-recruitment domain from a zebrafish guanylate-binding protein

Authors
Jin, T., Huang, M., Smith, P., Jiang, J., and Xiao, T.S.
ID
ZDB-PUB-130816-47
Date
2013
Source
Acta crystallographica. Section F, Structural biology and crystallization communications   69(8): 855-860 (Journal)
Registered Authors
Keywords
CARDs, six-helix bundle, zebrafish, guanylate-binding proteins
MeSH Terms
  • Amino Acid Sequence
  • Animals
  • CARD Signaling Adaptor Proteins/chemistry*
  • CARD Signaling Adaptor Proteins/genetics*
  • Crystallography, X-Ray
  • GTP-Binding Proteins/chemistry*
  • GTP-Binding Proteins/genetics*
  • Molecular Sequence Data
  • Neoplasm Proteins/chemistry*
  • Neoplasm Proteins/genetics*
  • Protein Structure, Secondary
  • Protein Structure, Tertiary
  • Zebrafish
  • Zebrafish Proteins/chemistry*
  • Zebrafish Proteins/genetics*
PubMed
23908027 Full text @ Acta Crystallogr. Sect. F Struct. Biol. Cryst. Commun.
Abstract

The caspase-recruitment domain (CARD) mediates homotypic protein-protein interactions that assemble large oligomeric signaling complexes such as the inflammasomes during innate immune responses. Structural studies of the mammalian CARDs demonstrate that their six-helix bundle folds belong to the death-domain superfamily, whereas such studies have not been reported for other organisms. Here, the zebrafish interferon-induced guanylate-binding protein 1 (zIGBP1) was identified that contains an N-terminal GTPase domain and a helical domain typical of the mammalian guanylate-binding proteins, followed by a FIIND domain and a C-terminal CARD similar to the mammalian inflammasome proteins NLRP1 and CARD8. The structure of the zIGBP1 CARD as a fusion with maltose-binding protein was determined at 1.47 Å resolution. This revealed a six-helix bundle fold similar to the NLRP1 CARD structure with the bent α1 helix typical of all known CARD structures. The zIGBP1 CARD surface contains a positively charged patch near its α1 and α4 helices and a negatively charged patch near its α2, α3 and α5 helices, which may mediate its interaction with partner domains. Further studies using binding assays and other analyses will be required in order to address the physiological function(s) of this zebrafish protein.

Genes / Markers
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Human Disease / Model
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Fish
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