PUBLICATION

Exome sequencing identifies NBEAL2 as the causative gene for gray platelet syndrome

Authors
Albers, C.A., Cvejic, A., Favier, R., Bouwmans, E.E., Alessi, M.C., Bertone, P., Jordan, G., Kettleborough, R.N., Kiddle, G., Kostadima, M., Read, R.J., Sipos, B., Sivapalaratnam, S., Smethurst, P.A., Stephens, J., Voss, K., Nurden, A., Rendon, A., Nurden, P., and Ouwehand, W.H.
ID
ZDB-PUB-110719-42
Date
2011
Source
Nature Genetics   43(8): 735-7 (Journal)
Registered Authors
Kettleborough, Ross
Keywords
none
MeSH Terms
  • Young Adult
  • Aged
  • Zebrafish/growth & development
  • Zebrafish/metabolism
  • Secretory Vesicles/metabolism*
  • Animals, Genetically Modified
  • Cytoplasmic Granules/metabolism*
  • Female
  • Nerve Tissue Proteins/antagonists & inhibitors
  • Nerve Tissue Proteins/genetics*
  • Gene Expression Regulation, Developmental
  • Blood Platelets/metabolism*
  • Blood Platelets/pathology
  • Humans
  • Adult
  • Base Sequence
  • Gray Platelet Syndrome/genetics*
  • Male
  • Sequence Analysis, DNA
  • Middle Aged
  • Pedigree
  • Animals
  • Molecular Sequence Data
  • Sequence Homology, Nucleic Acid
  • Embryo, Nonmammalian/cytology
  • Embryo, Nonmammalian/metabolism
PubMed
21765411 Full text @ Nat. Genet.
Abstract
Gray platelet syndrome (GPS) is a predominantly recessive platelet disorder that is characterized by mild thrombocytopenia with large platelets and a paucity of α-granules; these abnormalities cause mostly moderate but in rare cases severe bleeding. We sequenced the exomes of four unrelated individuals and identified NBEAL2 as the causative gene; it has no previously known function but is a member of a gene family that is involved in granule development. Silencing of nbeal2 in zebrafish abrogated thrombocyte formation.
Genes / Markers
Figures
No images available
Show all Figures
Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping