PUBLICATION

A mutation in the RNF170 gene causes autosomal dominant sensory ataxia

Authors
Valdmanis, P.N., Dupre, N., Lachance, M., Stochmanski, S.J., Belzil, V.V., Dion, P.A., Thiffault, I., Brais, B., Weston, L., Saint-Amant, L., Samuels, M.E., and Rouleau, G.A.
ID
ZDB-PUB-101201-45
Date
2011
Source
Brain : a journal of neurology   134(Pt 2): 602-607 (Journal)
Registered Authors
Saint-Amant, Louis
Keywords
ataxia, sensory neuropathy, gene identification, ring-finger protein, RNF170
MeSH Terms
  • Amino Acid Sequence
  • Animals
  • Ataxia/genetics*
  • Ataxia/metabolism
  • Cell Death/drug effects
  • DNA Copy Number Variations
  • Gene Knockdown Techniques/methods
  • Humans
  • Mutation, Missense*
  • Oligodeoxyribonucleotides, Antisense/pharmacology
  • Ubiquitin-Protein Ligases/genetics*
  • Ubiquitin-Protein Ligases/metabolism
  • Zebrafish
PubMed
21115467 Full text @ Brain
Abstract
Autosomal dominant sensory ataxia is a rare genetic condition that results in a progressive ataxia that is caused by degeneration of the posterior columns of the spinal cord. To date only two families have been clinically ascertained with this condition, both from Maritime Canada. We previously mapped both families to chromosome 8p12-8q12 and have now screened the majority of annotated protein-coding genes in the shared haplotype region by direct DNA sequencing. We have identified a putative pathogenic mutation in the gene encoding ring-finger protein RNF170, a potential ubiquitin ligase. This mutation is a rare non-synonymous change in a well-conserved residue and is predicted to be pathogenic by SIFT, PolyPhen, PANTHER and Align-GVD. Microinjection of wild-type or mutant orthologous messenger RNAs into zebrafish (Danio rerio) embryos confirmed that the mutation dominantly disrupts normal embryonic development. Together these results suggest that the mutation in RNF170 is causal for the sensory ataxia in these families.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping