PUBLICATION

Characterization of the AP-1 mu1A and mu1B adaptins in zebrafish (Danio rerio)

Authors
Zizioli, D., Forlanelli, E., Guarienti, M., Nicoli, S., Fanzani, A., Bresciani, R., Borsani, G., Preti, A., Cotelli, F., and Schu, P.
ID
ZDB-PUB-100726-21
Date
2010
Source
Developmental Dynamics : an official publication of the American Association of Anatomists   239(9): 2404-2412 (Journal)
Registered Authors
Borsani, Giuseppe, Cotelli, Franco, Nicoli, Stefania
Keywords
adaptor protein complexes, AP-1, clathrin, trans-Golgi network, membrane traffic, zebrafish, gut
MeSH Terms
  • Adaptor Protein Complex 1/genetics
  • Adaptor Protein Complex 1/metabolism*
  • Adaptor Protein Complex mu Subunits/classification
  • Adaptor Protein Complex mu Subunits/genetics
  • Adaptor Protein Complex mu Subunits/metabolism*
  • Amino Acid Sequence
  • Animals
  • Gene Expression Regulation, Developmental
  • Gene Knockdown Techniques
  • Genome
  • Humans
  • Mice
  • Molecular Sequence Data
  • Morphogenesis/physiology*
  • Phenotype
  • Phylogeny
  • Protein Isoforms/classification
  • Protein Isoforms/genetics
  • Protein Isoforms/metabolism*
  • Sequence Alignment
  • Tissue Distribution
  • Zebrafish/anatomy & histology
  • Zebrafish/embryology*
  • Zebrafish/genetics
  • Zebrafish/growth & development*
  • Zebrafish Proteins/classification
  • Zebrafish Proteins/genetics
  • Zebrafish Proteins/metabolism*
PubMed
20652956 Full text @ Dev. Dyn.
Abstract
Protein transport between the trans-Golgi network and endosomes is mediated by transport vesicles formed by the adaptor-protein complex AP-1, consisting of the adaptins gamma1, beta1, mu1, sigma1. Mammalia express mu1A ubiquitously and isoform mu1B in polarized epithelia. Mouse gamma1 or mu1A 'knock out's revealed that AP-1 is indispensable for embryonic development. We isolated mu1A and mu1B from Danio rerio. Analysis of mu1A and mu1B expression revealed tissue-specific expression for either one during embryogenesis and in adult tissues in contrast to their expression in mammalia. mu1B transcript was detected in organs of endodermal derivation and "knock-down" experiments gave rise to embryos defective in formation of intestine, liver, and pronephric ducts. Development ceased at 7-8 dpf. mu1B is not expressed in murine liver, indicating loss of mu1B expression and establishment of alternative sorting mechanisms during mammalian development.
Genes / Markers
Figures
Show all Figures
Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping