PUBLICATION

olig1 expression identifies developing oligodendrocytes in zebrafish and requires hedgehog and notch signaling

Authors
Schebesta, M., and Serluca, F.C.
ID
ZDB-PUB-090309-10
Date
2009
Source
Developmental Dynamics : an official publication of the American Association of Anatomists   238(4): 887-898 (Journal)
Registered Authors
Serluca, Fabrizio
Keywords
olig1, olig2, myelin, oligodendrocytes, zebrafish, hedgehog, notch, eGFP
MeSH Terms
  • Amino Acid Sequence
  • Animals
  • Animals, Genetically Modified
  • Brain/cytology
  • Brain/embryology
  • Brain/metabolism
  • Cell Differentiation
  • Conserved Sequence
  • Embryo, Nonmammalian/cytology
  • Embryo, Nonmammalian/embryology
  • Embryo, Nonmammalian/metabolism
  • Gene Expression Regulation, Developmental
  • Hedgehog Proteins/metabolism*
  • Humans
  • Molecular Sequence Data
  • Oligodendroglia/cytology*
  • Oligodendroglia/metabolism*
  • Phylogeny
  • Promoter Regions, Genetic/genetics
  • Receptors, Notch/metabolism*
  • Sequence Alignment
  • Signal Transduction
  • Spinal Cord/cytology
  • Spinal Cord/embryology
  • Spinal Cord/metabolism
  • Transcription Factors/chemistry
  • Transcription Factors/genetics
  • Transcription Factors/metabolism*
  • Zebrafish/embryology*
  • Zebrafish/genetics
  • Zebrafish/metabolism*
  • Zebrafish Proteins/chemistry
  • Zebrafish Proteins/genetics
  • Zebrafish Proteins/metabolism*
PubMed
19253391 Full text @ Dev. Dyn.
Abstract
Myelin, the isolating sheath around large diameter axons, is formed in the central nervous system (CNS) by oligodendrocytes. We isolated the zebrafish ortholog of olig1, a bHLH transcription factor, and describe the origin and development of oligodendrocytes in the zebrafish brain. Olig1:mem-eGFP transgenic animals demonstrate the highly dynamic nature of oligodendrocyte membrane processes, providing a tool for studying in vivo oligodendrocyte development. Formation of oligodendrocytes and initiation of olig1 expression are under the control of long-range hedgehog and notch signaling while maintenance of olig1 expression only depends on hedgehog. Over-expression of olig1 did not affect myelin formation in the brain and combined over-expression of olig1 and olig2 could not rescue loss of hedgehog signaling, indicating that critical factors other than olig1 and olig2 are necessary. Lastly, knockdown of Olig1 in an Olig2-sensitized background did result in defects in CNS myelination, indicating a functional overlap between Olig1 and Olig2 proteins.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping