PUBLICATION

Superagonistic fluorinated vitamin D(3) analogs stabilize helix 12 of the vitamin D receptor

Authors
Eelen, G., Valle, N., Sato, Y., Rochel, N., Verlinden, L., De Clercq, P., Moras, D., Bouillon, R., Muñoz, A., and Verstuyf, A.
ID
ZDB-PUB-081028-9
Date
2008
Source
Chemistry & Biology   15(10): 1029-1034 (Journal)
Registered Authors
Keywords
CHEMBIOL, SIGNALING
MeSH Terms
  • Protein Structure, Tertiary
  • Models, Molecular
  • Humans
  • Crystallography, X-Ray
  • beta Catenin/genetics
  • Cell Line, Tumor
  • TCF Transcription Factors/genetics
  • Protein Structure, Secondary
  • Binding Sites
  • Transcription, Genetic/drug effects
  • Transcription, Genetic/genetics
  • Receptors, Calcitriol/chemistry*
  • Receptors, Calcitriol/genetics
  • Receptors, Calcitriol/metabolism*
  • Cell Differentiation/drug effects
  • Cholecalciferol/agonists*
  • Cholecalciferol/analogs & derivatives*
  • Cholecalciferol/chemistry
  • Fluorine Compounds/agonists*
  • Fluorine Compounds/chemistry
PubMed
18940664 Full text @ Chem. Biol.
Abstract
Side chain fluorination is often used to make analogs of 1,25-dihydroxyvitamin D(3) [1,25(OH)(2)D(3)] resistant to degradation by 24-hydroxylase. The fluorinated nonsteroidal analogs CD578, WU515, and WY1113 have an increased prodifferentiating action on SW480-ADH colon cancer cells, which correlated with stronger induction of vitamin D receptor (VDR)-coactivator interactions and stronger repression of beta-catenin/TCF activity. Cocrystallization of analog CD578 with the zebrafish (z)VDR and an SRC-1 coactivator peptide showed that the fluorine atoms of CD578 make additional contacts with Val444 and Phe448 of activation helix 12 (H12) of the zVDR and with Leu440 of the H11-H12 loop. Consequently, the SRC-1 peptide makes more contacts with the VDR-CD578 complex than with the VDR-1,25(OH)(2)D(3) complex. These data show that fluorination not only affects degradation of an analog but can also have direct effects on H12 stabilization.
Genes / Markers
Figures
No images available
Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping