PUBLICATION

The human homologue of the yeast polyubiquitination factor Ufd2p is cleaved by caspase 6 and granzyme B during apoptosis

Authors
Mahoney, J.A., Odin, J.A., White, S.M., Shaffer, D., Koff, A., Casciola-Rosen, L., and Rosen, A.
ID
ZDB-PUB-020214-2
Date
2002
Source
The Biochemical journal   361(3): 587-595 (Journal)
Registered Authors
Keywords
none
MeSH Terms
  • Amino Acid Motifs
  • Amino Acid Sequence
  • Apoptosis*
  • Baculoviridae/metabolism
  • Binding Sites
  • Blotting, Northern
  • Caspase 6
  • Caspases/metabolism*
  • Cloning, Molecular
  • Enzyme Inhibitors/pharmacology
  • Fungal Proteins/chemistry*
  • Fungal Proteins/metabolism
  • Granzymes
  • HeLa Cells
  • Humans
  • Kinetics
  • Molecular Sequence Data
  • Mutagenesis, Site-Directed
  • Precipitin Tests
  • Protein Binding
  • Protein Structure, Tertiary
  • Recombinant Proteins/metabolism
  • Saccharomyces cerevisiae Proteins*
  • Sequence Homology, Amino Acid
  • Serine Endopeptidases/metabolism*
  • Staurosporine/pharmacology
  • Tissue Distribution
  • Ubiquitin/metabolism
  • Ubiquitin-Conjugating Enzymes
  • Ultraviolet Rays
PubMed
11802788 Full text @ Biochem. J.
Abstract
In the present study, we demonstrate that a human homologue of Ufd2p (a yeast protein that catalyses the formation of long polyubiquitin chains, and is implicated in responses to environmental stress), UFD2 (ubiquitin fusion degradation protein-2), is cleaved during apoptosis induced by multiple stimuli, including UVB irradiation, Fas ligation, staurosporine treatment and cytotoxic lymphocyte granule-induced death. Caspase 6 and granzyme B efficiently cleave UFD2 [k(cat)/K(m)=(4-5)x10(4) M(-1).s(-1)] at Asp(123), whereas caspases 3 and 7 cleave UFD2 approx. 10-fold less efficiently immediately upstream at Asp(109). Thus UFD2 is added to the growing list of proteins with closely spaced caspase and granzyme B cleavage sites, suggesting the presence of a previously unrecognized, conserved motif. Both cleavage sites are contained and conserved within a novel 300-amino-acid N-terminal domain present in apparent UFD2 orthologues in mice and zebrafish, but absent in all UFD2 family members in lower eukaryotes. Full-length recombinant UFD2 exhibited ubiquitin-protein ligase ('E3')-like ubiquitination activity in vitro, but this activity was abolished in recombinant UFD2 truncated at the granzyme B/caspase 6 cleavage site. Cleavage of UFD2 by caspases or granzyme B within this putative regulatory N-terminal domain might have important functional consequences within the apoptotic cascade.
Genes / Markers
Figures
Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping