ZFIN Person: Shin, Jordan

ZFIN ID: ZDB-PERS-030925-8

Shin, Jordan

Email:	jshin1@partners.org
URL:
Affiliation:
Address:	Cardiovascular Research Center Massachusetts General Hospital Mailcode 1494100 149 13th Street, 4th Floor Charlestown, MA 02129 USA
Country:
Phone:	(617) 724-9566
Fax:	(617) 726-5806
ORCID ID:

BIOGRAPHY AND RESEARCH INTERESTS

PUBLICATIONS

Martins Metelo, A., Noonan, H.R., Xiang, L., Jin, Y., Baker, R., Kamentsky, L., Zhang, Y., van Rooijen, E., Shin, J., Carpenter, A.E., Yeh, J.R., Peterson, R.T., Iliopoulos, O. (2015) Pharmacological HIF2α inhibition improves VHL disease–associated phenotypes in zebrafish model. J. Clin. Invest.. 125(5):1987-97

Liu, Y., Asnani, A., Zou, L., Bentley, V.L., Yu, M., Wang, Y., Dellaire, G., Sarkar, K.S., Dai, M., Chen, H.H., Sosnovik, D.E., Shin, J.T., Haber, D.A., Berman, J.N., Chao, W., Peterson, R.T. (2014) Visnagin protects against doxorubicin-induced cardiomyopathy through modulation of mitochondrial malate dehydrogenase. Science Translational Medicine. 6:266ra170

Becker, J.R., Chatterjee, S., Robinson, T.Y., Bennett, J.S., Panáková, D., Galindo, C.L., Zhong, L., Shin, J.T., Coy, S.M., Kelly, A.E., Roden, D.M., Lim, C.C., and MacRae, C.A. (2014) Differential activation of natriuretic peptide receptors modulates cardiomyocyte proliferation during development. Development (Cambridge, England). 141(2):335-345

Shin, J.T., Ward, J.E., Collins, P.A., Dai, M., Semigran, H.L., Semigran, M.J., and Seldin, D.C. (2012) Overexpression of human amyloidogenic light chains causes heart failure in embryonic zebrafish: a preliminary report. Amyloid : the international journal of experimental and clinical investigation : the official journal of the International Society of Amyloidosis. 19(4):191-196

Chong, N.W., Koekemoer, A.L., Ounzain, S., Samani, N.J., Shin, J.T., and Shaw, S.Y. (2012) STARS Is Essential to Maintain Cardiac Development and Function In Vivo via a SRF Pathway. PLoS One. 7(7):e40966

Shin, J.T., Pomerantsev, E.V., Mably, J.D., and MacRae, C.A. (2010) High-resolution cardiovascular function confirms functional orthology of myocardial contractility pathways in zebrafish. Physiological Genomics. 42(2):300-309

Wang, L., Sewell, W.F., Kim, S.D., Shin, J.T., Macrae, C.A., Zon, L.I., Seidman, J.G., and Seidman, C.E. (2008) Eya4 regulation of Na+/K+-ATPase is required for sensory system development in zebrafish. Development (Cambridge, England). 135(20):3425-3434

Heuser, A., Plovie, E.R., Ellinor, P.T., Grossmann, K.S., Shin, J.T., Wichter, T., Basson, C.T., Lerman, B.B., Sasse-Klaassen, S., Thierfelder, L., MacRae, C.A., and Gerull, B. (2006) Mutant desmocollin-2 causes arrhythmogenic right ventricular cardiomyopathy. American journal of human genetics. 79(6):1081-1088

Shin, J.T., Priest, J.R., Ovcharenko, I., Ronco, A., Moore, R.K., Burns, C.G., and MacRae, C.A. (2005) Human-zebrafish non-coding conserved elements act in vivo to regulate transcription. Nucleic acids research. 33(17):5437-45

Schonberger, J., Wang, L., Shin, J.T., Kim, S.D., Depreux, F.F., Zhu, H., Zon, L., Pizard, A., Kim, J.B., MacRae, C.A., Mungall, A.J., Seidman, J.G., and Seidman, C.E. (2005) Mutation in the transcriptional coactivator EYA4 causes dilated cardiomyopathy and sensorineural hearing loss. Nature Genetics. 37(4):418-422

Shin, J.T. and Fishman, M.C. (2002) From zebrafish to human: modular medical models. Annual review of genomics and human genetics. 3:311-340

NON-ZEBRAFISH PUBLICATIONS