ZFIN ID: ZDB-PERS-001109-4
Geling, Andrea
Email: ageling@web.de
URL:
Affiliation:
Address: GSF-Research Center Institute of Mammalian Genetics Ingolstaedter Landstrasse 1 Neuherberg, D-85758 Germany
Country:
Phone: 49-89-3187-2887
Fax: 49-89-3187-3099
Orcid ID:


BIOGRAPHY AND RESEARCH INTERESTS


PUBLICATIONS
Kikuta, H., Laplante, M., Navratilova, P., Komisarczuk, A.Z., Engstrom, P.G., Fredman, D., Akalin, A., Caccamo, M., Sealy, I., Howe, K., Ghislain, J., Pezeron, G., Mourrain, P., Ellingsen, S., Oates, A.C., Thisse, C., Thisse, B., Foucher, I., Adolf, B., Geling, A., Lenhard, B., and Becker, T.S. (2007) Genomic regulatory blocks encompass multiple neighboring genes and maintain conserved synteny in vertebrates. Genome research. 17(5):545-555
Stigloher, C., Ninkovic, J., Laplante, M., Geling, A., Tannhauser, B., Topp, S., Kikuta, H., Becker, T.S., Houart, C., and Bally-Cuif, L. (2006) Segregation of telencephalic and eye-field identities inside the zebrafish forebrain territory is controlled by Rx3. Development (Cambridge, England). 133(15):2925-2935
Geling, A., Plessy, C., Rastegar, S., Strähle, U., and Bally-Cuif, L. (2004) Her5 acts as a prepattern factor that blocks neurogenin1 and coe2 expression upstream of Notch to inhibit neurogenesis at the midbrain-hindbrain boundary. Development (Cambridge, England). 131(9):1993-2006
Geling, A. (2004) Functional analysis of the bHLH transcription factor Her5 during midbrain-hindbrain neurogenesis in the embryonic zebrafish. Ph.D. Thesis.
Geling, A., Itoh, M., Tallafuss, A., Chapouton, P., Tannhauser, B., Kuwada, J.Y., Chitnis, A.B., and Bally-Cuif, L. (2003) bHLH transcription factor Her5 links patterning to regional inhibition of neurogenesis at the midbrain-hindbrain boundary. Development (Cambridge, England). 130(8):1591-1604
Geling, A., Steiner, H., Willem, M., Bally-Cuif, L., and Haass, C. (2002) A {gamma}-secretase inhibitor blocks Notch signaling in vivo and causes a severe neurogenic phenotype in zebrafish. EMBO reports. 3(7):688-694

NON-ZEBRAFISH PUBLICATIONS