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Fig. 1
The antiviral effect of compound 4 against MNV and the HuNoV GI.1 replicon. (A) The antiviral activity of compound 4 against MNV was quantified by (i) virus-induced CPE reduction and (ii) reduction in viral RNA levels. Cellular toxicity was assessed in parallel. n = 3–4 (±SEM). (B) The antiviral activity of compound 4 against the HuNoV GI.1 replicon. Intracellular RNA loads are represented by relative genome quantification of the HuNoV GI.1 replicon versus β-actin. n = 5 (±SEM).
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