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Fig. 5
Forced expression of Pax1 decreases cell death potential on physiological stress. U2OS cells were exposed to 80 J/m2 UV (a) or 2.5 µM doxorubicin (b) for 8 h. Hoechst-positive cells were counted and subjected to statistical analysis using Student’s -tests. All pax1b DNA induction was mediated by the IRES-TOMATO lentivirus system. Data are presented as means ± SE from three independent experiments. *P < 0.05, compared with vector.
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