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Fig. S5
Cyp26a1 deficiency does not alter the ability of RA sensor cells to respond. (A1-3, B1-3) Tg(β-actin:GDBD-RLBD);(UAS:EGFP) donor cells into either WT or Cyp26a1 deficient hosts injected with mrfp mRNA have similar levels of GFP expression in the notochord and spinal cord compared to WT siblings.
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Reprinted from Developmental Biology, 405(1), Rydeen, A., Voisin, N., D'Aniello, E., Ravisankar, P., Devignes, C.S., Waxman, J.S., Excessive feedback of Cyp26a1 promotes cell non-autonomous loss of retinoic acid signaling, 47-55, Copyright (2015) with permission from Elsevier. Full text @ Dev. Biol.