Image
|
Figure Caption
Fig. S2 Enc1l-MO2 and enc1l-MOsp can affect LR organ asymmetry.(A): enc1l-MO1 efficiently blocks the expression of Enc1l-GFP fusion constructs injected into zebrafish embryos at the 1-cell stage. (B): RT-PCR shows that enc1l-MOsp causes abnormal reduction of enc1l-mRNA but not the internal β-actin mRNA control. (C): Injection of enc1l-MO2 or enc1l-MOsp disrupts the pancreas LR asymmetry. (D): Injection of enc1l-MO2 disrupts the heart LR asymmetry.
Acknowledgments
This image is the copyrighted work of the attributed author or publisher, and
ZFIN has permission only to display this image to its users.
Additional permissions should be obtained from the applicable author or publisher of the image.
Reprinted from Developmental Biology, 374(1), Qian, M., Yao, S., Jing, L., He, J., Xiao, C., Zhang, T., Meng, W., Zhu, H., Xu, H., and Mo, X., ENC1-like Integrates the Retinoic Acid/FGF Signaling Pathways to Modulate Ciliogenesis of Kupffer's Vesicle during Zebrafish Embryonic Development, 85-95, Copyright (2013) with permission from Elsevier. Full text @ Dev. Biol.