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Fig. S4 Posterolateral specification of the inner ear is not dependent on AP hindbrain patterning. (A-C) krox20 expression in embryos treated from 10.5 until 24 hpf with DMSO (A), 20 nM RA (B) or 20 μM DEAB (C), showing no hindbrain patterning defects. (D-G) Expression of krox20 (D,E) and tbx1 (F,G) in wild-type and valb337 mutant embryos. Note the strong reduction of krox20 expression in rhombomere 5 (E). Anterior is to the left.
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