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Fig. S4 Pbx-depleted and meis1-morphant embryos exhibit normal posterior hox gene expression. Shown are representative embryos following in situ hybridization analyses of hoxb6b (A, B, G, H), hoxb7a (C, D, I, J), and hoxa9a (E, F, K, L) expression in wild type (WT; A, C, E, G, I, K), Pbx-depleted (B, D, F), and meis1-morphant (H, J, L) embryos. The PLM of 16 hpf flat-mounted, deyolked embryos is shown in dorsal view with anterior oriented to the left. In comparison to WT embryos, Pbx-depleted and meis1-morphant embryos exhibit normal hoxb6b, hoxb7a, and hoxa9a expression.
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Reprinted from Developmental Biology, 340(2), Pillay, L.M., Forrester, A.M., Erickson, T., Berman, J.N., and Waskiewicz, A.J., The Hox cofactors Meis1 and Pbx act upstream of gata1 to regulate primitive hematopoiesis, 306-317, Copyright (2010) with permission from Elsevier. Full text @ Dev. Biol.