Phenotype
|
Conditions
|
Figures
|
anatomical system quality, abnormal
|
standard conditions
|
Fig. for (tu29a)
from Phenotype Annotation (1994-2006)
|
apoptotic process increased occurrence, abnormal
|
standard conditions
|
Fig. 7
from Kwak et al., 2006
|
blood circulation disrupted, abnormal
|
standard conditions
|
text only
from Brand et al., 1996
|
brain lacks all parts of type midbrain hindbrain boundary, abnormal
|
standard conditions
|
Fig. 4
from Porreca et al., 2012
Fig. 7
from Pfeffer et al., 1998
|
brain quality, abnormal
|
standard conditions
|
Fig. for (tu29a)
from Phenotype Annotation (1994-2006)
|
caudal commissure increased size, abnormal
|
standard conditions
|
Fig. 2
from Scholpp et al., 2003
|
cerebellum absent, abnormal
|
standard conditions
|
Fig. 1
from Lun et al., 1998
text only
from Brand et al., 1996
|
closure of optic fissure decreased process quality, abnormal
|
chemical treatment by environment: Z-Val-Ala-Asp(OMe)-CH2F
|
Fig. 4
from Gregory-Evans et al., 2011
|
closure of optic fissure decreased process quality, abnormal
|
control
|
Fig. 4
from Gregory-Evans et al., 2011
|
commissura cerebelli aplastic, abnormal
|
standard conditions
|
Fig. 1
from Scholpp et al., 2003
|
cranial ganglion neuron decreased amount, abnormal
|
standard conditions
|
Fig. S1
from Nechiporuk et al., 2007
|
cranial nerve II malformed, abnormal
|
standard conditions
|
Fig. 4
from Macdonald et al., 1997
|
cranial nerve IV aplastic, abnormal
|
standard conditions
|
Fig. 1
from Scholpp et al., 2003
|
diencephalon distended, abnormal
|
standard conditions
|
text only
from Scholpp et al., 2003
|
epiphysis decreased width, abnormal
|
standard conditions
|
Fig. 2
from Scholpp et al., 2003
|
eye decreased size, abnormal
|
standard conditions
|
Fig. 7
from Moosajee et al., 2008
|
eye anatomical region vegfaa expression decreased amount, abnormal
|
standard conditions
|
Figure 3
from Weaver et al., 2020
|
eye anatomical region vegfc expression decreased amount, abnormal
|
standard conditions
|
Figure 3
from Weaver et al., 2020
|
eye anatomical region vegfab expression decreased amount, abnormal
|
standard conditions
|
Figure 3
from Weaver et al., 2020
|
eye apoptotic process increased occurrence, abnormal
|
control
|
Fig. 4
from Gregory-Evans et al., 2011
|
eye apoptotic process occurrence, ameliorated
|
chemical treatment by environment: Z-Val-Ala-Asp(OMe)-CH2F
|
Fig. 4
from Gregory-Evans et al., 2011
|
eye apoptotic process occurrence, ameliorated
|
chemical treatment by environment: curcumin
|
Fig. 4
from Gregory-Evans et al., 2011
|
gut epithelium edematous, abnormal
|
standard conditions
|
Fig. 1
from Lun et al., 1998
|
head flt4 expression decreased amount, abnormal
|
standard conditions
|
Figure 2
from Weaver et al., 2020
|
head vegfab expression decreased amount, abnormal
|
standard conditions
|
Figure 3
from Weaver et al., 2020
|
head cldn5b expression decreased amount, abnormal
|
standard conditions
|
Figure 2
from Weaver et al., 2020
|
head vegfaa expression decreased amount, abnormal
|
standard conditions
|
Figure 3
from Weaver et al., 2020
|
head vegfc expression decreased amount, abnormal
|
standard conditions
|
Figure 3
from Weaver et al., 2020
|
head tjp1a expression decreased amount, abnormal
|
standard conditions
|
Figure 2
from Weaver et al., 2020
|
head tln1 expression decreased amount, abnormal
|
standard conditions
|
Figure 2
from Weaver et al., 2020
|
heart edematous, abnormal
|
standard conditions
|
Fig. 5
from Shima et al., 2009
|
hindbrain distended, abnormal
|
standard conditions
|
text only
from Scholpp et al., 2003
|
lens decreased size, abnormal
|
standard conditions
|
Fig. 7
from Moosajee et al., 2008
|
lens opaque, abnormal
|
standard conditions
|
Fig. 7
from Moosajee et al., 2008
|
locus ceruleus development disrupted, abnormal
|
standard conditions
|
Fig. 6
from Guo et al., 1999
|
locus coeruleus absent, abnormal
|
standard conditions
|
Fig. 6
from Guo et al., 1999
|
midbrain hindbrain boundary absent, abnormal
|
standard conditions
|
Fig. 1
from Lun et al., 1998
text only
from Brand et al., 1996
Table 2
from Chen et al., 1996
|
midbrain hindbrain boundary aplastic, abnormal
|
standard conditions
|
Fig. 1
from Scholpp et al., 2003
|
midbrain hindbrain boundary tln1 expression decreased amount, abnormal
|
standard conditions
|
Figure 2
from Weaver et al., 2020
|
midbrain hindbrain boundary physical object quality, abnormal
|
standard conditions
|
Fig. 9
from Pfeffer et al., 1998
Fig. 9
from Reifers et al., 1998
|
midbrain hindbrain boundary physical object quality, normal
|
standard conditions
|
Fig. 9
from Reifers et al., 1998
|
midbrain hindbrain boundary quality, abnormal
|
standard conditions
|
Fig. for (tu29a)
from Phenotype Annotation (1994-2006)
|
midbrain hindbrain boundary neural keel physical object quality, abnormal
|
standard conditions
|
Fig. 8
from Pfeffer et al., 1998
|
midbrain-hindbrain boundary development arrested, abnormal
|
standard conditions
|
Fig. 1
from Lun et al., 1998
|
midbrain-hindbrain boundary development decreased process quality, abnormal
|
standard conditions
|
Fig. 9
from Reifers et al., 1998
|
midbrain-hindbrain boundary development process quality, normal
|
standard conditions
|
Fig. 9
from Reifers et al., 1998
|
midbrain-hindbrain boundary initiation process quality, abnormal
|
standard conditions
|
Fig. 7
from Pfeffer et al., 1998
|
nervous system quality, abnormal
|
standard conditions
|
Fig. for (tu29a)
from Phenotype Annotation (1994-2006)
|
optic chiasm cranial nerve II ipsilateral to optic chiasm cranial nerve II, abnormal
|
standard conditions
|
Fig. 5
from Shima et al., 2009
|
optic cup physical object quality, abnormal
|
standard conditions
|
Fig. 9
from Pfeffer et al., 1998
|
optic fissure tln1 expression decreased amount, abnormal
|
standard conditions
|
Figure 2
from Weaver et al., 2020
|
optic fissure increased width, abnormal
|
control
|
Fig. 4
from Gregory-Evans et al., 2011
|
optic fissure increased width, abnormal
|
chemical treatment by environment: Z-Val-Ala-Asp(OMe)-CH2F
|
Fig. 4
from Gregory-Evans et al., 2011
|
optic fissure increased width, exacerbated
|
chemical treatment by environment: curcumin
|
Fig. 4
from Gregory-Evans et al., 2011
|
optic furrow mmp2 expression decreased amount, abnormal
|
standard conditions
|
Figure 5
from Weaver et al., 2020
|
optic furrow mmp14b expression decreased amount, abnormal
|
standard conditions
|
Figure 5
from Weaver et al., 2020
|
optic furrow mmp14a expression decreased amount, abnormal
|
standard conditions
|
Figure 5
from Weaver et al., 2020
|
optic furrow basement membrane increased amount, abnormal
|
standard conditions
|
Figure 1
from Weaver et al., 2020
|
optic furrow filamentous actin decreased amount, abnormal
|
standard conditions
|
Figure 1
from Weaver et al., 2020
|
optic furrow laminin complex increased amount, abnormal
|
standard conditions
|
Figure 1
from Weaver et al., 2020
|
optic nerve development disrupted, abnormal
|
standard conditions
|
Fig. 4
from Macdonald et al., 1997
|
optic tectum absent, abnormal
|
standard conditions
|
Fig. 1
from Lun et al., 1998
text only
from Brand et al., 1996
|
optic tectum aplastic, abnormal
|
standard conditions
|
Fig. 1
from Scholpp et al., 2003
|
optic tectum apoptotic, abnormal
|
standard conditions
|
Fig. 2
from Brand et al., 1996
|
optic tectum decreased size, abnormal
|
standard conditions
|
Table 1
from Trowe et al., 1996
|
optic tectum morphology, abnormal
|
standard conditions
|
Table 1
from Trowe et al., 1996
|
otic vesicle decreased size, abnormal
|
standard conditions
|
Fig. 5
from Shima et al., 2009
text only
from Brand et al., 1996
|
otic vesicle morphology, normal
|
standard conditions
|
Fig. 5
from Mackereth et al., 2005
|
otic vesicle physical object quality, abnormal
|
standard conditions
|
Fig. 8
from Pfeffer et al., 1998
|
otic vesicle neuroblast delamination increased occurrence, abnormal
|
standard conditions
|
Fig. 4
from Kantarci et al., 2016
|
otic vesicle neuroblast proliferation decreased occurrence, abnormal
|
standard conditions
|
Fig. 4
from Kantarci et al., 2016
|
otic vesicle ventro-lateral region gsc expression increased distribution, abnormal
|
standard conditions
|
Fig. 4
from Kantarci et al., 2016
|
parasympathetic nervous system development disrupted, abnormal
|
standard conditions
|
Fig. S1
from Nechiporuk et al., 2007
|
pericardium edematous, abnormal
|
standard conditions
|
Fig. 7
from Moosajee et al., 2008
Fig. 1
from Lun et al., 1998
|
pronephric duct absent, abnormal
|
standard conditions
|
text only
from Brand et al., 1996
|
pronephric duct lacks all parts of type epithelial cell brush border, abnormal
|
standard conditions
|
Fig. 6
from Majumdar et al., 2000
|
pronephric duct physical object quality, abnormal
|
standard conditions
|
Fig. 9
from Pfeffer et al., 1998
|
pronephric duct central region distended, abnormal
|
standard conditions
|
Fig. 6
from Majumdar et al., 2000
|
pronephric duct epithelial cell flattened, abnormal
|
standard conditions
|
Fig. 6
from Majumdar et al., 2000
|
pronephros absent, abnormal
|
standard conditions
|
Table 2
from Chen et al., 1996
|
pronephros physical object quality, abnormal
|
standard conditions
|
Fig. 9
from Pfeffer et al., 1998
|
pronephros quality, abnormal
|
standard conditions
|
Fig. for (tu29a)
from Phenotype Annotation (1994-2006)
|
pronephros development process quality, abnormal
|
standard conditions
|
Fig. 1
from Majumdar et al., 2000
|
retina layer formation disrupted, abnormal
|
standard conditions
|
Fig. 7
from Moosajee et al., 2008
|
retinal ganglion cell axon guidance process quality, abnormal
|
standard conditions
|
Table 1
from Trowe et al., 1996
|
retinal ganglion cell axon guidance quality, abnormal
|
standard conditions
|
Fig. for (tu29a)
from Phenotype Annotation (1994-2006)
|
retinal neural layer closure incomplete, abnormal
|
standard conditions
|
Fig. 3
from Macdonald et al., 1997
|
retinal pigmented epithelium closure incomplete, abnormal
|
standard conditions
|
Fig. 3
from Macdonald et al., 1997
|
swim bladder aplastic, abnormal
|
standard conditions
|
Fig. 7
from Moosajee et al., 2008
|
thyroid follicle aplastic, abnormal
|
standard conditions
|
Fig. 4
from Wendl et al., 2002
|
utricle lacks parts or has fewer parts of type hair cell, abnormal
|
standard conditions
|
Fig. 7
from Kwak et al., 2006
|
whole organism decreased length, abnormal
|
standard conditions
|
Fig. 7
from Moosajee et al., 2008
|
whole organism lethal (sensu genetics), abnormal
|
standard conditions
|
Fig. for (tu29a)
from Phenotype Annotation (1994-2006)
|
whole organism viability, abnormal
|
standard conditions
|
Table 1
from Trowe et al., 1996
|
whole organism apoptotic process increased occurrence, abnormal
|
control
|
Fig. 4
from Gregory-Evans et al., 2011
|
whole organism apoptotic process occurrence, ameliorated
|
chemical treatment by environment: curcumin
|
Fig. 4
from Gregory-Evans et al., 2011
|
whole organism apoptotic process occurrence, ameliorated
|
chemical treatment by environment: Z-Val-Ala-Asp(OMe)-CH2F
|
Fig. 4
from Gregory-Evans et al., 2011
|