Morpholino

MO1-slc39a5

ID

ZDB-MRPHLNO-210604-1

Name

MO1-slc39a5

Previous Names

None

Target

slc39a5 (1)

Sequence

5' - AGACTGTCTGTCCACCCATGATTTT - 3'

Disclaimer

Although ZFIN verifies reagent sequence data, we recommend that you conduct independent sequence analysis before ordering any reagent.

Note

Translation-blocking MO.

Genome Resources

None

Target Location

Genomic Features

No data available

Expression

Gene expression in Wild Types + MO1-slc39a5

Expressed Gene	Anatomy	Figures
hbae3	caudal vein plexus	Fig. 1 from Xia et al., 2020

Phenotype

Phenotype resulting from MO1-slc39a5

Phenotype	Fish	Figures
blood vessel endothelial cell filopodium decreased amount, abnormal	cz3325Tg; is5Tg + MO1-slc39a5	Fig. 2 from Xia et al., 2020
blood vessel endothelial cell filopodium increased amount, abnormal	cz3325Tg; is5Tg + MO1-slc39a5	Fig. 2 from Xia et al., 2020
caudal vein plexus hbae3 expression increased amount, abnormal	cz3325Tg + MO1-slc39a5	Fig. 1 from Xia et al., 2020
caudal vein plexus capillary decreased amount, abnormal	cz3325Tg; is5Tg + MO1-slc39a5	Fig. 2 from Xia et al., 2020
caudal vein plexus endothelial cell migration disrupted, abnormal	y1Tg + MO1-slc39a5	Fig. 3 from Xia et al., 2020
caudal vein plexus endothelial cell proliferation disrupted, abnormal	y1Tg + MO1-slc39a5	Fig. 3 from Xia et al., 2020
caudal vein plexus nucleate erythrocyte increased amount, abnormal	cz3325Tg + MO1-slc39a5	Fig. 1 from Xia et al., 2020
heart blood circulation decreased efficacy, abnormal	WT + MO1-slc39a5	Fig. 1 from Xia et al., 2020
intracellular zinc ion homeostasis disrupted, abnormal	WT + MO1-slc39a5	Fig. 5 from Xia et al., 2020
sprouting angiogenesis delayed, abnormal	cz3325Tg; is5Tg + MO1-slc39a5	Fig. 2 from Xia et al., 2020
subintestinal venous plexus sprouting angiogenesis disrupted, abnormal	y1Tg + MO1-slc39a5	Fig. S2 from Xia et al., 2020
whole organism zinc atom increased amount, abnormal	WT + MO1-slc39a5	Fig. 5 from Xia et al., 2020

Phenotype of all Fish created by or utilizing MO1-slc39a5

Phenotype	Fish	Conditions	Figures
whole organism zinc atom increased amount, abnormal	WT + MO1-slc39a5	standard conditions	Fig. 5 from Xia et al., 2020
heart blood circulation decreased efficacy, abnormal	WT + MO1-slc39a5	standard conditions	Fig. 1 from Xia et al., 2020
intracellular zinc ion homeostasis disrupted, abnormal	WT + MO1-slc39a5	standard conditions	Fig. 5 from Xia et al., 2020
caudal vein plexus nucleate erythrocyte increased amount, abnormal	cz3325Tg + MO1-slc39a5	standard conditions	Fig. 1 from Xia et al., 2020
caudal vein plexus hbae3 expression increased amount, abnormal	cz3325Tg + MO1-slc39a5	standard conditions	Fig. 1 from Xia et al., 2020
caudal vein plexus endothelial cell proliferation disrupted, abnormal	y1Tg + MO1-slc39a5	standard conditions	Fig. 3 from Xia et al., 2020
caudal vein plexus capillary amount, ameliorated	y1Tg + MO1-slc39a5	chemical treatment by environment: N,N,N',N'-tetrakis(2-pyridylmethyl)ethylenediamine	Fig. 5 from Xia et al., 2020
caudal vein plexus endothelial cell migration disrupted, abnormal	y1Tg + MO1-slc39a5	standard conditions	Fig. 3 from Xia et al., 2020
blood vessel endothelial cell filopodium amount, ameliorated	y1Tg + MO1-slc39a5	chemical treatment by environment: N,N,N',N'-tetrakis(2-pyridylmethyl)ethylenediamine	Fig. 5 from Xia et al., 2020
subintestinal venous plexus sprouting angiogenesis disrupted, abnormal	y1Tg + MO1-slc39a5	standard conditions	Fig. S2 from Xia et al., 2020
sprouting angiogenesis delayed, abnormal	cz3325Tg; is5Tg + MO1-slc39a5	standard conditions	Fig. 2 from Xia et al., 2020
blood vessel endothelial cell filopodium increased amount, abnormal	cz3325Tg; is5Tg + MO1-slc39a5	standard conditions	Fig. 2 from Xia et al., 2020
blood vessel endothelial cell filopodium decreased amount, abnormal	cz3325Tg; is5Tg + MO1-slc39a5	standard conditions	Fig. 2 from Xia et al., 2020
caudal vein plexus capillary decreased amount, abnormal	cz3325Tg; is5Tg + MO1-slc39a5	standard conditions	Fig. 2 from Xia et al., 2020

Citations