Rose et al., 2020 - SCO-Spondin Defects and Neuroinflammation Are Conserved Mechanisms Driving Spinal Deformity across Genetic Models of Idiopathic Scoliosis. Current biology : CB   30(12):2363-2373.e6 Full text @ Curr. Biol.
Human Disease / Model Data (1 Record)
Human Disease Fish Environment Evidence Code
idiopathic scoliosis sspodmh4/+
odad3ts272/ts272
ptk7ahsc9/hsc9 		standard conditions
standard conditions
standard conditions

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