HUMAN DISEASE MODELED by y1Tg + MO1-amotl2a
No data available

GENE EXPRESSION
Gene expression in y1Tg + MO1-amotl2a

PHENOTYPE
Phenotype in y1Tg + MO1-amotl2a

Phenotype	Conditions	Figures
angiogenesis disrupted, abnormal	standard conditions	Fig. 1 from Wang et al., 2011
blood vessel unlumenized, abnormal	standard conditions	Fig. 1 from Wang et al., 2011
dorsal aorta constricted, abnormal	standard conditions	Fig. 2 from Hultin et al., 2014
dorsal aorta decreased diameter, abnormal	standard conditions	Fig. 2 from Hultin et al., 2014
dorsal aorta blood vessel lumenization decreased process quality, abnormal	standard conditions	Fig. 2 from Hultin et al., 2014
dorsal aorta morphogenesis decreased process quality, abnormal	standard conditions	Fig. 2 from Hultin et al., 2014
dorsal longitudinal anastomotic vessel aplastic, abnormal	standard conditions	Fig. 1 from Wang et al., 2011
intersegmental vessel decreased length, abnormal	standard conditions	Fig. 1 from Wang et al., 2011
posterior cardinal vein diameter, normal	standard conditions	Fig. 2 from Hultin et al., 2014
posterior cardinal vein morphology, normal	standard conditions	Fig. 2 from Hultin et al., 2014
posterior cardinal vein open, normal	standard conditions	Fig. 2 from Hultin et al., 2014
posterior cardinal vein blood vessel lumenization process quality, normal	standard conditions	Fig. 2 from Hultin et al., 2014
protein phosphorylation decreased occurrence, abnormal	standard conditions	Fig. 5 from Wang et al., 2011
whole organism lacks all parts of type parachordal vessel, abnormal	standard conditions	Fig. 1 from Wang et al., 2011