ZFIN ID: ZDB-FISH-150901-16962
Fish name: noton1/n1(AB)
Genotype: noton1/n1 (AB)
Targeting Reagent: none

HUMAN DISEASE MODELED by noton1/n1(AB)
No data available
GENE EXPRESSION
Gene expression in noton1/n1(AB)
RNA expression
Expressed Gene Structure Conditions Figures
alcama standard conditions Fig. 5 with image from Schilling et al., 1999
ascl1a standard conditions text only from Cau et al., 2003
atf7a standard conditions Fig. 5 with image from Zhao et al., 2005
bmp4 standard conditions Fig. 5 with image from Schilling et al., 1999
Fig. 3 with image from Chen et al., 1997
chrd standard conditions Fig. 5 with image from Miller-Bertoglio et al., 1997
dnah9 standard conditions text only from Essner et al., 2005
emx1 standard conditions text only from Kawahara et al., 2002
foxa2 standard conditions Fig. 6 with image from Bisgrove et al., 2000
gdf6b standard conditions text only from Bruneau et al., 1997
gsc standard conditions Fig. 6 with image from Bisgrove et al., 2000
isl1 standard conditions text only from Cau et al., 2003
kctd12.1 standard conditions text only from Gamse et al., 2003
kdrl standard conditions Fig. 3 with image from Majumdar et al., 2000
mdka standard conditions Fig. 5 with image from Schäfer et al., 2005
myod1 standard conditions Fig. 6 with image from Groves et al., 2005
Fig. 11 with image from Schier et al., 1997
Fig. 5 with image from Halpern et al., 1995
chemical treatment: SU5402 Fig. 6 with image from Groves et al., 2005
ndr1 standard conditions text only from Essner et al., 2005
ndr2 standard conditions Fig. 5 with image from Rebagliati et al., 1998
neurod1 standard conditions text only from Cau et al., 2003
neurog1 standard conditions text only from Cau et al., 2003
noto standard conditions text only from Cau et al., 2003
otx5 standard conditions text only from Gamse et al., 2003
pax2a standard conditions Fig. 6 with image from Bisgrove et al., 2000
pax9 standard conditions Fig. 2 with image from Majumdar et al., 2000
shha standard conditions Fig. 4 with image from Schäfer et al., 2005
Fig. 9 with imageFig. 10 with image from Schier et al., 1997
snai1a standard conditions Fig. 5 with imageFig. 6 with image from Halpern et al., 1995
tbxta standard conditions text only from Essner et al., 2005
Fig. 6 with image from Bisgrove et al., 2000
Fig. 6 with image from Halpern et al., 1995
tie1 standard conditions Fig. 3 with image from Majumdar et al., 2000
twist2 standard conditions Fig. 2 with image from Majumdar et al., 2000
Fig. 4 with image from Halpern et al., 1995
vegfaa standard conditions Fig. 3 with image from Majumdar et al., 2000
wnt4b standard conditions Fig. 3 from Liu et al., 2000
wt1a standard conditions Fig. 1 with image from Majumdar et al., 2000
Protein expression
Antibody Antigen Genes Structure Conditions Figures
zn-5 alcama standard conditions Fig. 5 with image from Schilling et al., 1999
Reporter gene expression No data available
PHENOTYPE
Phenotype in noton1/n1(AB)
Phenotype Conditions Figures
axial chorda mesoderm cell fate commitment disrupted, abnormal standard conditions Fig. 3 with image from Halpern et al., 1995
axial mesoderm structural organization disrupted, abnormal standard conditions Fig. 6 with image from Halpern et al., 1995
cell migration involved in kidney development disrupted, abnormal standard conditions Fig. 1 with imageFig. 3 with imageFig. 5 with image from Majumdar et al., 2000
determination of digestive tract left/right asymmetry decreased process quality, abnormal standard conditions Fig. 5 with image from Schilling et al., 1999
determination of heart left/right asymmetry decreased process quality, abnormal standard conditions Fig. 5 with image from Schilling et al., 1999
floor plate midbrain region whole, normal standard conditions Fig. 1 with image from Halpern et al., 1995
floor plate rhombomere region whole, normal standard conditions Fig. 1 with image from Halpern et al., 1995
floor plate spinal cord region patchy, abnormal standard conditions Fig. 1 with image from Halpern et al., 1995
glomerular filtration decreased process quality, abnormal standard conditions Fig. 4 with image from Majumdar et al., 2000
glomerular filtration present, normal standard conditions Fig. 4 with image from Majumdar et al., 2000
heart tube bilateral symmetry, abnormal standard conditions Fig. 5 with image from Schilling et al., 1999
intestinal bulb primordium bilateral symmetry, abnormal standard conditions Fig. 5 with image from Schilling et al., 1999
muscle displaced to axis, abnormal standard conditions Fig. 1 with image from Halpern et al., 1995
notochord absent, abnormal standard conditions Fig. 1 with image from Halpern et al., 1995
notochord aplastic, abnormal standard conditions Table 1 from Lewis et al., 2001
notochord aplastic/hypoplastic, abnormal standard conditions Fig. 8 with image from Schier et al., 1997
notochord quality, abnormal standard conditions Fig. for (n1) from Phenotype Annotation (1994-2006)
notochord cell differentiation disrupted, abnormal standard conditions Fig. 1 with imageFig. 3 with image from Halpern et al., 1995
notochord cell differentiation process quality, abnormal standard conditions Fig. 7 with image from Melby et al., 1996
notochord development disrupted, abnormal standard conditions Fig. 1 with image from Halpern et al., 1995
podocyte mislocalised laterally, abnormal standard conditions Fig. 1 with imageFig. 2 with image from Majumdar et al., 2000
podocyte morphology, normal standard conditions Fig. 3 with imageFig. 5 with image from Majumdar et al., 2000
podocyte present, normal standard conditions Fig. 1 with imageFig. 2 with image from Majumdar et al., 2000
primary motor neuron decreased amount, abnormal standard conditions Table 1 from Lewis et al., 2001
pronephric capsular space present, normal standard conditions Fig. 5 with image from Majumdar et al., 2000
pronephric glomerular basement membrane morphology, normal standard conditions Fig. 5 with image from Majumdar et al., 2000
pronephric glomerular basement membrane lamina densa morphology, normal standard conditions Fig. 5 with image from Majumdar et al., 2000
pronephric glomerular capillary attached to common cardinal vein, abnormal standard conditions Fig. 5 with image from Majumdar et al., 2000
pronephric glomerular capillary blood circulation present, normal standard conditions Fig. 5 with image from Majumdar et al., 2000
pronephric glomerular capillary endothelial cell present, normal standard conditions Fig. 3 with image from Majumdar et al., 2000
pronephric glomerular capillary glomerular endothelium fenestra closed, abnormal standard conditions Fig. 5 with image from Majumdar et al., 2000
pronephric glomerular capillary membrane detached from pronephric glomerular basement membrane, abnormal standard conditions Fig. 5 with image from Majumdar et al., 2000
pronephric glomerulus increased amount, abnormal standard conditions Fig. 1 with imageFig. 3 with imageFig. 5 with image from Majumdar et al., 2000
pronephric glomerulus malformed, abnormal standard conditions Fig. 5 with image from Majumdar et al., 2000
pronephric glomerulus mislocalised laterally, abnormal standard conditions Fig. 1 with imageFig. 3 with imageFig. 5 with image from Majumdar et al., 2000
sclerotome absent, abnormal standard conditions Fig. 2 with image from Majumdar et al., 2000
somite fused with somite, abnormal standard conditions Fig. 1 with image from Halpern et al., 1995
somite malformed, abnormal standard conditions Fig. 8 with image from Schier et al., 1997
somite quality, abnormal standard conditions Fig. for (n1) from Phenotype Annotation (1994-2006)
somite shape, abnormal standard conditions Table 1 from Lewis et al., 2001
somite development process quality, abnormal standard conditions Fig. 1 with imageFig. 3 with image from Halpern et al., 1995
whole organism edematous, abnormal standard conditions Fig. 4 with image from Majumdar et al., 2000
whole organism lethal (sensu genetics), abnormal standard conditions Fig. for (n1) from Phenotype Annotation (1994-2006)
whole organism viability, abnormal standard conditions Table 1 from Lewis et al., 2001

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