ZFIN ID: ZDB-FIG-130321-6
Chen et al., 2013 - Functional characterization of Klippel-Trenaunay syndrome gene AGGF1 identifies a novel angiogenic signaling pathway for specification of vein differentiation during embryogenesis. Human molecular genetics   22(5):963-976 Full text @ Hum. Mol. Genet.
ADDITIONAL FIGURES
EXPRESSION / LABELING:
Genes:
Fish:
Knockdown Reagents:
Anatomical Terms:
Stage: Prim-5
PHENOTYPE:
Fish:
Knockdown Reagent:
Observed In:
Stage Range: Prim-5 to Long-pec

Fig. 5 ZFIN is incorporating published figure images and captions as part of an ongoing project. Figures from some publications have not yet been curated, or are not available for display because of copyright restrictions.

Gene Expression Details
Gene Antibody Fish Conditions Stage Qualifier Anatomy Assay
dab2 AB control Prim-5 posterior cardinal vein ISH
AB + MO1-aggf1 standard conditions Prim-5 Not Detected posterior cardinal vein ISH
AB + MO2-aggf1 standard conditions Prim-5 posterior cardinal vein ISH
efnb2a AB control Prim-5 dorsal aorta ISH
AB + MO1-aggf1 standard conditions Prim-5 dorsal aorta ISH
ephb4a AB control Prim-5 posterior cardinal vein ISH
AB + MO1-aggf1 standard conditions Prim-5 Not Detected posterior cardinal vein ISH
flt4 AB control Prim-5 posterior cardinal vein ISH
AB + MO1-aggf1 standard conditions Prim-5 Not Detected posterior cardinal vein ISH
AB + MO2-aggf1 standard conditions Prim-5 posterior cardinal vein ISH
hey2 AB control Prim-5 dorsal aorta ISH
AB + MO1-aggf1 standard conditions Prim-5 dorsal aorta ISH
AB + MO2-aggf1 standard conditions Prim-5 dorsal aorta ISH
notch3 AB control Prim-5 dorsal aorta ISH
AB + MO1-aggf1 standard conditions Prim-5 dorsal aorta ISH
AB + MO2-aggf1 standard conditions Prim-5 dorsal aorta ISH
Antibody Labeling Details No data available
Phenotype Details
Fish Conditions Stage Phenotype
la116Tg + MO1-aggf1 standard conditions Prim-5 dorsal aorta present, normal
Prim-5 posterior cardinal vein absent, abnormal
Long-pec dorsal aorta present, normal
Long-pec posterior cardinal vein absent, abnormal
Full text @ Hum. Mol. Genet.